[Endothelial dysfunction as a pathogenetic factor of sensorineural hearing loss].

Q3 Medicine
V V Noskova, A N Khrabrikov
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引用次数: 0

Abstract

Chronic sensorineural hearing loss (SNHL) is a common disease that leads to disability of the population. Despite the many reports devoted to SNHL, the question of the pathogenesis of the disease is still open. Many researchers consider the development of SNHL as a manifestation of microangiopathy. The mechanism of development of microangiopathy in SNHL is multifactorial, but most researchers agree that endothelial dysfunction (ED) triggers a complex of pathological changes in the vessels of the inner ear.

Objective: Review of the results of scientific research in recent years on the problem of etiopathogenesis of sensorineural hearing loss from the perspective of endothelial dysfunction in the formation of auditory disorders.

[内皮功能障碍是感音神经性听力损失的致病因素之一]。
慢性感音神经性听力损失(SNHL)是一种导致人口残疾的常见疾病。尽管有许多关于 SNHL 的报道,但该病的发病机理问题仍然悬而未决。许多研究人员认为,SNHL 的发生是微血管病变的一种表现。SNHL微血管病的发病机制是多因素的,但大多数研究人员都认为内皮功能障碍(ED)引发了内耳血管的复杂病理变化:从内皮功能障碍在听觉障碍形成过程中的作用角度,回顾近年来关于感音神经性听力损失病因发病机制问题的科学研究成果。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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来源期刊
Vestnik otorinolaringologii
Vestnik otorinolaringologii Medicine-Otorhinolaryngology
CiteScore
0.80
自引率
0.00%
发文量
69
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