{"title":"Dr. Jong Hoon Kim","authors":"None Jong Hoon Kim","doi":"10.2336/nishinihonhifu.85.397","DOIUrl":null,"url":null,"abstract":"Dr. Jong Hoon Kim M.D. Ph.D. is an Assistant Professor in the Department of Dermatology and Cutaneous Biology Research Institute at Yonsei University in South Korea. He completed his Ph.D. and postdoctoral projects between 2013 and 2016, which focused on the study of T cell immunology. His initial research aimed to dissect the mechanism of T-cell activation in psoriasis. Through his work, he found that TCR-mediated activation of PD-1+IL-17A+ T cells contributes to disease activity in murine psoriasis, independent of IL-23-mediated activation (J Allergy Clin Immunol. 2016). This study provided insights into the mechanism of generating PD-1+ resident memory Tc17 cells observed in human psoriatic skin. He also conducted a study on human T cells by collecting liver perfusate during liver transplantation, where he identified specific features of liverresident CD69+CD103-CD8+ T cells under healthy and diseased conditions (J Hepatol. 2020).","PeriodicalId":19437,"journal":{"name":"Nishi Nihon Hifuka","volume":"36 1","pages":"0"},"PeriodicalIF":0.0000,"publicationDate":"2023-10-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":null,"platform":"Semanticscholar","paperid":null,"PeriodicalName":"Nishi Nihon Hifuka","FirstCategoryId":"1085","ListUrlMain":"https://doi.org/10.2336/nishinihonhifu.85.397","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
引用次数: 0
Abstract
Dr. Jong Hoon Kim M.D. Ph.D. is an Assistant Professor in the Department of Dermatology and Cutaneous Biology Research Institute at Yonsei University in South Korea. He completed his Ph.D. and postdoctoral projects between 2013 and 2016, which focused on the study of T cell immunology. His initial research aimed to dissect the mechanism of T-cell activation in psoriasis. Through his work, he found that TCR-mediated activation of PD-1+IL-17A+ T cells contributes to disease activity in murine psoriasis, independent of IL-23-mediated activation (J Allergy Clin Immunol. 2016). This study provided insights into the mechanism of generating PD-1+ resident memory Tc17 cells observed in human psoriatic skin. He also conducted a study on human T cells by collecting liver perfusate during liver transplantation, where he identified specific features of liverresident CD69+CD103-CD8+ T cells under healthy and diseased conditions (J Hepatol. 2020).