The Effect of Repeated Electromagnetic Fields Stimulation in Biological Systems

Felipe P. Perez, J. Rizkalla, M. Jeffers, P. Salama, Cristina N. Perez Chumbiauca, M. Rizkalla
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引用次数: 2

Abstract

The effects of electromagnetic fields on living organs have been explored with the use of both biological experimentation and computer simulations. In this paper we will examine the effects of the repeated electromagnetic field stimulation (REMFS) on cell cultures, mouse models, and computer simulations for diagnostic purposes. In our biological experiments we used 50 MHz and 64 MHz since this is approved in MRI systems. REMFS upregulated pathways that control the aging process such as proteostasis. REMFS delayed and reversed cellular senescence in mouse and human cell cultures. More recently we determined that REMFS decreases toxic protein beta amyloid levels, which is the cause of Alzheimer’s disease (AD), in human neuronal cultures. The mechanism of these effects is the reactivation of the heat shock factor 1 (HSF1). HSF1 activation is a quantum effect of the EMF-oscillations on the water that surrounds a long non-coding RNA, allowing it to then bind and activate the HSF1. We also performed electromagnetic (EM) computer simulations of virtual prototypes of bone cancer, femur fracture, and diabetic foot ulcers utilizing different frequencies and power applications to build an accurate differential diagnosis. These applications indicate the feasibility of subsequent practical models for diagnosing and treating human diseases.
重复电磁场刺激在生物系统中的作用
电磁场对活体器官的影响已经通过生物实验和计算机模拟进行了探索。在本文中,我们将研究重复电磁场刺激(REMFS)对细胞培养、小鼠模型和用于诊断目的的计算机模拟的影响。在我们的生物实验中,我们使用了50 MHz和64 MHz,因为这在MRI系统中是被批准的。REMFS上调了控制衰老过程的途径,如蛋白质停滞。在小鼠和人类细胞培养中,REMFS延缓和逆转了细胞衰老。最近,我们确定REMFS降低了人类神经元培养中导致阿尔茨海默病(AD)的毒性蛋白-淀粉样蛋白水平。这些作用的机制是热休克因子1 (HSF1)的再激活。HSF1的激活是围绕着长链非编码RNA的水的电磁场振荡的量子效应,允许它结合并激活HSF1。我们还利用不同的频率和功率应用对骨癌、股骨骨折和糖尿病足溃疡的虚拟原型进行了电磁(EM)计算机模拟,以建立准确的鉴别诊断。这些应用表明了诊断和治疗人类疾病的后续实用模型的可行性。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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