Bilateral Ischemic Lumbosacral Plexopathy after Proximal Aortic Surgery

Min Gi Kim, Seung Hak Lee
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Abstract

In paraplegia after proximal aortic surgery, in addition to spinal cord ischemia, injuries to the plexuses and other peripheral nerves should be considered. Spinal cord ischemia—the most common etiology of paraplegia—can be caused by the occlusion of several radicular arteries resulting from aortic clamping. We report a case of bilateral ischemic lumbosacral plexopathy with a spared spinal cord occurring after proximal aortic surgery. A 76-year-old woman underwent aortic valve replacement with ascending aorta and total arch replacement, along with multiple hematoma evacuations. Postoperatively, she developed paraplegia with sensory deficits in the bilateral lower limbs. Spinal magnetic resonance imaging revealed no remarkable findings at any level of the spinal cord. In electrophysiological studies, the compound motor action potential and sensory nerve action potential of the bilateral lower extremities did not respond to stimuli; all examined muscles displayed abnormal spontaneous activities without motor unit action potentials. Based on these findings, a diagnosis of bilateral lumbosacral plexopathy was ultimately made.
近端主动脉手术后双侧缺血性腰骶神经丛病
主动脉近端手术后截瘫,除脊髓缺血外,还应考虑神经丛等周围神经的损伤。脊髓缺血是截瘫最常见的病因,可由主动脉夹持导致的几根动脉闭塞引起。我们报告一例在主动脉近端手术后发生的双侧缺血性腰骶神经丛病伴保留脊髓。一位76岁的女性接受了升主动脉主动脉瓣置换术和全弓置换术,同时进行了多次血肿清除。术后,患者出现双侧下肢感觉缺陷截瘫。脊髓核磁共振成像显示在脊髓的任何水平没有显著的发现。电生理研究发现,双侧下肢的复合运动动作电位和感觉神经动作电位对刺激无反应;所有被检查的肌肉都表现出异常的自发活动,没有运动单位动作电位。基于这些发现,最终诊断为双侧腰骶神经丛病。
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