The Causes of Logoneurosis in Childhood

Ye. P. Kharchenko, I. Zavadska
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Abstract

The purpose of our research is to develop the problem of stuttering in the psychological aspect to reveal its genesis, to understand the behavior of people who stutter in the process of communication, to identify their individual psycho-logical characteristics.research methods. The research methods were the method of observation and the empirical study of patients. The place of organizing the empirical stage of our research was the psychiatric hospital No 1 in Kyiv. With the help of clinical, pathopsychological and anamnestic methods 86 children at the age 3­12 years old (the average age is 8 ± 0.5 years old) with logoneurosis in anamnesis were diagnosed.the results of the research. The pathogenesis of stuttering is studied and those who stuttered are noted to have various autonomic changes. 84% of peo-ple who stuttered are considered to have autonomic dystonia. Among 98% of people who suffered from logoneurosis, 20% had a high intracranial pressure and extrapyramidal disorders. It is studied that those people who stuttered were born with vasoneurotics. It was quite objectively showed a change in the neu-rovegetative response in those ones who stuttered during seizures: in 100% of cases there was dilation of the pupils (mydriasis), while in a case of people who spoke correctly, the width of the pupils during speech did not change or there was some narrowing (miosis).conclusions. The reasons of logoneurosis are: neuropathic burden of par-ents (nervous, infectious and somatic diseases that weaken or disrupt the func-tions of the central nervous system); neuropathic features of a stuttering person (night terrors, enuresis, irritability, emotional tension); constitutional predisposi-tion (diseases of the autonomic nervous system and increased susceptibility to higher nervous activity, its special predisposition to mental trauma); hereditary burden; brain damage in different periods of the personal development.
儿童耳根神经症的病因
我们的研究目的是在心理方面发展口吃问题,揭示其成因,了解口吃者在交际过程中的行为,识别他们的个体心理特征。研究方法。研究方法为观察法和患者实证研究法。我们研究的实证阶段是在基辅的第一精神病院进行的。采用临床、病理心理及记忆方法对86例3 ~ 12岁(平均年龄8±0.5岁)的遗忘性颈神经官能症患儿进行诊断。研究的结果。研究了口吃的发病机制,发现口吃者有各种自主神经变化。84%的口吃者被认为患有自主神经张力障碍。在98%的患者中,20%的患者有高颅内压和锥体外系疾病。据研究,口吃的人生来就患有血管神经症。相当客观地显示了癫痫发作期间口吃患者神经植物反应的变化:100%的病例中瞳孔扩张(瞳孔缩小),而在说话正确的病例中,瞳孔宽度没有改变或有一些缩小(瞳孔缩小)。神经症的原因是:父母的神经性负担(神经、传染性和躯体疾病,削弱或破坏中枢神经系统的功能);口吃者的神经病变特征(夜惊、遗尿、易怒、情绪紧张);体质倾向(自主神经系统疾病和对高神经活动的易感性增加,其对精神创伤的特殊倾向);世袭的负担;个体发展不同时期的脑损伤。
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