Influence of abiotic stress on biomolecules

K. Zhanassova, A. Kurmanbayeva, Z. Masalimov
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Abstract

Plants are exposed continuously in their natural environment to diverse abiotic assaults throughout life, which leads to a decrease in yield. As a result, plants have evolved unique mechanisms at the morphological, physiological, and biochemical levels to help them resist, adapt, or survive severe environmental circumstances. Unfavorable environmental effects cause disruptions such as the inhibition of growth and photosynthesis, the closing of stomata, discoloration, and the accumulation of toxic forms of ROS in plants. However, currently, a substantial quantity of information has been gathered on the role of reactive oxygen species (ROS). Sunlight, temperature, salt content, heavy metals, air and water pollution, and UV radiation are all known to contribute to the production of reactive oxygen species (ROS) in plants. ROS harm plant systems structurally, causing a disruption in their equilibrium and the development of oxidative stress. Without appropriate control, the quantity of ROS in cells rises, causing oxidative damage to cell membranes, proteins, RNA and DNA molecules, and even cell death. It is critical to understand how plants respond to various stimuli and develop resistance to them. The review article aims to provide a general overview of the impact of oxidative stress on biomolecules in response to different abiotic stresses.
非生物胁迫对生物分子的影响
植物一生在自然环境中不断受到各种非生物攻击,导致产量下降。因此,植物在形态、生理和生化水平上进化出了独特的机制,以帮助它们抵抗、适应或生存恶劣的环境。不利的环境影响会导致植物生长和光合作用的抑制、气孔关闭、变色以及有毒ROS形式的积累等破坏。然而,目前已经收集了大量关于活性氧(ROS)作用的信息。众所周知,阳光、温度、盐含量、重金属、空气和水污染以及紫外线辐射都有助于植物体内活性氧(ROS)的产生。活性氧从结构上危害植物系统,引起植物系统平衡的破坏和氧化应激的发展。如果没有适当的控制,细胞中ROS的数量会上升,导致细胞膜、蛋白质、RNA和DNA分子的氧化损伤,甚至导致细胞死亡。了解植物如何对各种刺激作出反应并产生抗性是至关重要的。本文综述了氧化应激对生物分子响应不同非生物应激的影响。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
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