{"title":"Neurogenic stuttering as a manifestation of stroke and a mask of dysphonia.","authors":"P R Rao","doi":"","DOIUrl":null,"url":null,"abstract":"<p><p>R. L. was a 52-year-old man who was referred for an SLP consultation to determine the nature of his fluency disorder, whether or not treatment would be beneficial, and finally whether resumption of pre-trauma vocational status was feasible. The patient was involved in a motor vehicle accident with no resulting detectable trauma. However, shortly after the accident, R. L. developed a severe dysfluency that was later described as cortical stuttering. We reviewed the medical and rehabilitation work-up that attempted to determine whether the communication disorder was functional or organic in origin. Once the fluency disorder was determined to be caused by a suspected small, focal, hemispheric lesion, a five-month treatment program was undertaken that used a noval prosthetic approach to restore fluency. Once fluency was restored with the use of an artificial larynx, a residual anomia was detected and treated. The case of R. L. illustrates a stuttering that appeared to be caused by a combined neurogenic dyspraxic (vocal control), dysarthric (motor control), and dysnomic (word-finding) dysfluency. The literature on this issue was reviewed and the underlying mechanism of recovery was discussed.</p>","PeriodicalId":77075,"journal":{"name":"Clinics in communication disorders","volume":"1 1","pages":"31-7"},"PeriodicalIF":0.0000,"publicationDate":"1991-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":null,"platform":"Semanticscholar","paperid":null,"PeriodicalName":"Clinics in communication disorders","FirstCategoryId":"1085","ListUrlMain":"","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
引用次数: 0
Abstract
R. L. was a 52-year-old man who was referred for an SLP consultation to determine the nature of his fluency disorder, whether or not treatment would be beneficial, and finally whether resumption of pre-trauma vocational status was feasible. The patient was involved in a motor vehicle accident with no resulting detectable trauma. However, shortly after the accident, R. L. developed a severe dysfluency that was later described as cortical stuttering. We reviewed the medical and rehabilitation work-up that attempted to determine whether the communication disorder was functional or organic in origin. Once the fluency disorder was determined to be caused by a suspected small, focal, hemispheric lesion, a five-month treatment program was undertaken that used a noval prosthetic approach to restore fluency. Once fluency was restored with the use of an artificial larynx, a residual anomia was detected and treated. The case of R. L. illustrates a stuttering that appeared to be caused by a combined neurogenic dyspraxic (vocal control), dysarthric (motor control), and dysnomic (word-finding) dysfluency. The literature on this issue was reviewed and the underlying mechanism of recovery was discussed.
R. L.是一名52岁的男性,他被转介到SLP咨询,以确定他的流利障碍的性质,治疗是否有益,最后是否恢复创伤前的职业状态是可行的。患者卷入了一场机动车辆事故,没有造成可检测到的创伤。然而,事故发生后不久,r.l.出现了严重的语言障碍,后来被描述为皮质性口吃。我们回顾了医疗和康复工作,试图确定沟通障碍是功能性的还是器质性的。一旦确定流畅性障碍是由疑似小的局灶性半球病变引起的,就进行了为期5个月的治疗计划,使用新型假体方法来恢复流畅性。一旦使用人工喉头恢复流利,就会发现并治疗残余的失语症。R. L.的病例说明了一种口吃,似乎是由神经源性运动障碍(声音控制)、运动障碍(运动控制)和语言障碍(找词)引起的。综述了国内外有关文献,并对其恢复机制进行了探讨。