Is spasticity in acquired brain injury a sensory problem?

Abstract

There is clinical and physiological evidence that presynaptic inhibition and sensory afferent pathways have an effect on what is considered a primarily upper motor neurone problem. This could redefine spasticity and the need to refocus assessment and management of spasticity is proposed with an emphasis on functional assessment and treatments which modulate sensory afferent thresholds in order to improve the precision of skilled movements.