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Oxidative Medicine and Cellular Longevity

SCI期刊

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2767

中科院SCI期刊分区 WOS期刊分区历年发表投稿信息同类期刊

中文名称：

氧化医学和细胞寿命

期刊缩写：

Oxid. Med. Cell. Longevity

ISSN：

print: 1942-0900
on-line: 1942-0994

研究领域：

生物-细胞生物学

创刊年份：

2008年

h-index：

Gold OA文章占比：

99.12%

原创研究文献占比：

77.83%

SCI收录类型：

Science Citation Index Expanded (SCIE) || Scopus (CiteScore) || Directory of Open Access Journals (DOAJ)

期刊官网：

https://www.hindawi.com/journals/omcl

期刊介绍英文：

Oxidative Medicine and Cellular Longevity is a unique peer-reviewed, Open Access journal that publishes original research and review articles dealing with the cellular and molecular mechanisms of oxidative stress in the nervous system and related organ systems in relation to aging, immune function, vascular biology, metabolism, cellular survival and cellular longevity. Oxidative stress impacts almost all acute and chronic progressive disorders and on a cellular basis is intimately linked to aging, cardiovascular disease, cancer, immune function, metabolism and neurodegeneration. The journal fills a significant void in today’s scientific literature and serves as an international forum for the scientific community worldwide to translate pioneering “bench to bedside” research into clinical strategies.

CiteScore：

CiteScore	SJR	SNIP	CiteScore排名
13.2	1.477	1.233	学科排名百分位大类：Biochemistry, Genetics and Molecular Biology 小类：Biochemistry 33 / 438 92% 大类：Biochemistry, Genetics and Molecular Biology 小类：Aging 4 / 38 90% 大类：Biochemistry, Genetics and Molecular Biology 小类：Cell Biology 39 / 285 86%

发文信息

中科院SCI期刊分区

大类	小类	TOP期刊	综述期刊
2区生物学	2区细胞生物学 CELL BIOLOGY	否	否

WOS期刊分区

历年影响因子

历年发表

2012年	164
2013年	256
2014年	211
2015年	300
2016年	642
2017年	617
2018年	699
2019年	871
2020年	861
2021年	1038
2022年	1274

投稿信息

出版周期：

Quarterly

出版语言：

English

出版国家(地区)：

UNITED STATES

审稿时长：

3-8 weeks

投稿网址：

http://mts.hindawi.com/login/

出版商：

Hindawi Publishing Corporation

编辑部地址：

LANDES BIOSCIENCE, 1002 WEST AVENUE, 2ND FLOOR, AUSTIN, USA, TX, 78701

PubMed链接：

http://www.ncbi.nlm.nih.gov/nlmcatalog?term=1942-0900%5BISSN%5D

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