急性肺水肿与NSTEMI。

Ashley Pilgrim
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Cardiogenic pulmonary edema is characterized by acute dyspnea caused by the accumulation of fluid within the lung's interstitial and/or alveolar spaces, which is the result of acutely elevated cardiac filling pressures.2 Noncardiogenic pulmonary edema is characterized by fluid accumulation within the alveolar space in the absence of elevated pulmonary capillary wedge pressure.2 These patients often present critically ill, and rapid identification and aggressive management is paramount in caring for patients with pulmonary edema. Dyspnea is the most common presentation with a sensitivity of 89% but a low specificity of 51%.3 Workup of pulmonary edema often includes laboratory testing, electrocardiogram (EKG), chest x-ray (CXR), and often bedside ultrasound (US) and echocardiography.4 Pulmonary edema management depends on the etiology but is often focused on preload and afterload reduction. Diuretics, nitrates, and optimizing ventilatory support through non-invasive and invasive strategies are the mainstay of treatment.</p><p><strong>Educational objectives: </strong>At the end of this practice oral boards case, the learner will:1) recognize unstable vital signs (VS) and intervene to stabilize ventilation and oxygenation, 2) demonstrate the ability to obtain a complete medical history including the important characteristics of chest pain, 3) demonstrate an appropriate exam on a patient, 4) order the appropriate evaluation studies for a patient with complaints of dyspnea, 5) interpret the results of diagnostic evaluation and diagnose Non- ST elevation myocardial infarction (NSTEMI) and pulmonary edema, 6) order appropriate management of pulmonary edema and NSTEMI, and 6) demonstrate effective communication with patient and family members.</p><p><strong>Educational methods: </strong>Practice oral boards.</p><p><strong>Research methods: </strong>Immediate Feedback was solicited from the learners and observers participating in the case both by verbal discussion and completion of a rating for the case following the debriefing. 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The differential for respiratory distress is large, but most learners were able to quickly identify pulmonary edema based on the exam findings of jugular vein distention (JVD), rales, and lower extremity edema. Most learners quickly escalated to a non-rebreather mask and ultimately to BPAP (bilevel positive airway pressure) without requesting to intubate the patient. There was notable variation in the approach to administering nitrates, but most ordered an intravenous (IV) nitroglycerin (NTG) drip and requested pharmacy assistance in dosing. Diuretics were ordered by all the learners, but some were hesitant to start early because they felt the effect would be delayed. Some of the residents did not identify ischemic changes on the EKG at first glance but did request to review a second time when the troponin result was positive. All residents gave aspirin after noting the positive troponin, but not all were able to make a clear diagnosis of NSTEMI or consult cardiology. Although the case was relatively straightforward, residents enjoyed early diagnosis and aggressive management of the patient with impending respiratory failure. Many residents are asking for an ultrasound early in the workup of this patient presenting in respiratory distress. Although not a critical action in this case, it highlights the emphasis placed on ultrasonography in the current emergency medicine curriculum.</p><p><strong>Topics: </strong>Pulmonary Edema, Cardiovascular emergencies, NSTEMI.</p>","PeriodicalId":73721,"journal":{"name":"Journal of education & teaching in emergency medicine","volume":"8 3","pages":"O1-O32"},"PeriodicalIF":0.0000,"publicationDate":"2023-07-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10414976/pdf/","citationCount":"1","resultStr":"{\"title\":\"Acute Pulmonary Edema and NSTEMI.\",\"authors\":\"Ashley Pilgrim\",\"doi\":\"10.21980/J8CW67\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"<p><strong>Audience: </strong>Emergency medicine residents and medical students on emergency medicine rotation.</p><p><strong>Introduction: </strong>Acute pulmonary edema is a common and potentially fatal presentation in the emergency department. 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Dyspnea is the most common presentation with a sensitivity of 89% but a low specificity of 51%.3 Workup of pulmonary edema often includes laboratory testing, electrocardiogram (EKG), chest x-ray (CXR), and often bedside ultrasound (US) and echocardiography.4 Pulmonary edema management depends on the etiology but is often focused on preload and afterload reduction. 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引用次数: 1

摘要

听众:急诊医学住院医师和急诊医学轮转的医学生。简介:急性肺水肿是急诊科常见且可能致命的症状。每年有超过100万的患者被诊断为继发于心脏原因的肺水肿肺水肿大致分为两大类:心源性和非心源性。心源性肺水肿的特征是肺间质和/或肺泡间隙内积液引起的急性呼吸困难,这是心脏充盈压力急性升高的结果非心源性肺水肿的特征是在肺毛细血管楔压未升高的情况下肺泡腔内积液这些患者通常表现为危重症,快速识别和积极治疗对肺水肿患者的护理至关重要。呼吸困难是最常见的表现,其敏感性为89%,但特异性较低,为51%肺水肿的检查通常包括实验室检查、心电图(EKG)、胸片(CXR),通常还有床边超声(US)和超声心动图肺水肿的管理取决于病因,但通常侧重于负荷前和负荷后的减少。利尿剂、硝酸盐和通过无创和有创策略优化通气支持是治疗的主要手段。教育目标:在这个练习口述板案例结束时,学习者将:1)识别不稳定的生命体征(VS)并进行干预以稳定通气和氧合,2)证明有能力获得完整的病史,包括胸痛的重要特征,3)证明对患者进行适当的检查,4)对抱怨呼吸困难的患者进行适当的评估研究。5)解释诊断评估结果并诊断非ST段抬高型心肌梗死(NSTEMI)和肺水肿,6)对肺水肿和NSTEMI进行适当的管理,6)与患者和家属进行有效的沟通。教学方法:练习口语。研究方法:通过口头讨论和在汇报后完成对案例的评分,向参与案例的学习者和观察者征求即时反馈。通过比较不同培训年份住院医师的评分指标来评估教育内容的有效性。美国研究生医学教育学院(ACGME)核心能力的评分采用1- 8,1 -4为不可接受的表现,5 - 8为可接受的表现。有效性是基于作为实践口头委员会候选人的住院医生完全完成病例,然后进行述职会议,讨论病例的关键组成部分。结果:本病例在一个相对较新的住院医师项目中被介绍给12名急诊医学住院医师,其中7名为PGY 1, 5名为PGY 2。居民的总体平均得分为5.62分。在这种情况下,PGY 1居民的平均得分为5.56,PGY 2居民的平均得分略好,为5.70。PGY 2住院医师的轻微改善可能归因于更多的临床经验;然而,在这次模拟体验之前,这两个班级都没有任何事先接触口语板形式的机会。6名住院医师完成了病例中所有关键动作。在那些错过关键行动的人中,未能诊断出NSTEMI和咨询心脏病学是最常见的。所有学习者都认为该案例具有教育价值,总体评分为4.83(1-5李克特量表,5为优秀)。讨论:急性肺水肿和NSTEMI是大多数急诊医生经常遇到的常见诊断。这个病例强调需要早期识别和积极的管理病人出现呼吸窘迫。呼吸窘迫的区别很大,但大多数学习者能够根据颈静脉扩张(JVD)、罗音和下肢水肿的检查结果快速识别肺水肿。大多数学习者很快升级到非呼吸面罩,并最终在没有要求插管的情况下升级到BPAP(双水平气道正压)。在施用硝酸盐的方法上有明显的变化,但大多数人要求静脉滴注硝酸甘油(NTG),并要求药房协助给药。所有的学习者都订购了利尿剂,但有些人犹豫要不要早点开始,因为他们觉得效果会延迟。有些住院医生在第一眼心电图上没有发现缺血性改变,但当肌钙蛋白结果呈阳性时,他们确实要求复查第二次。所有居民在注意到肌钙蛋白阳性后都服用阿司匹林,但并非所有人都能明确诊断为非stemi或咨询心脏病学。 虽然病例相对简单,但住院医生对即将发生呼吸衰竭的患者进行了早期诊断和积极的治疗。许多住院医师都要求在这个病人出现呼吸窘迫的早期检查中进行超声检查。虽然在这种情况下不是一个关键的行动,它突出强调超声检查在当前急诊医学课程。主题:肺水肿,心血管急症,NSTEMI。
本文章由计算机程序翻译,如有差异,请以英文原文为准。

Acute Pulmonary Edema and NSTEMI.

Acute Pulmonary Edema and NSTEMI.

Acute Pulmonary Edema and NSTEMI.

Acute Pulmonary Edema and NSTEMI.

Audience: Emergency medicine residents and medical students on emergency medicine rotation.

Introduction: Acute pulmonary edema is a common and potentially fatal presentation in the emergency department. More than 1 million patients are admitted annually with a diagnosis of pulmonary edema secondary to cardiac causes.1 Pulmonary edema is broadly split into two main categories: cardiogenic and noncardiogenic. Cardiogenic pulmonary edema is characterized by acute dyspnea caused by the accumulation of fluid within the lung's interstitial and/or alveolar spaces, which is the result of acutely elevated cardiac filling pressures.2 Noncardiogenic pulmonary edema is characterized by fluid accumulation within the alveolar space in the absence of elevated pulmonary capillary wedge pressure.2 These patients often present critically ill, and rapid identification and aggressive management is paramount in caring for patients with pulmonary edema. Dyspnea is the most common presentation with a sensitivity of 89% but a low specificity of 51%.3 Workup of pulmonary edema often includes laboratory testing, electrocardiogram (EKG), chest x-ray (CXR), and often bedside ultrasound (US) and echocardiography.4 Pulmonary edema management depends on the etiology but is often focused on preload and afterload reduction. Diuretics, nitrates, and optimizing ventilatory support through non-invasive and invasive strategies are the mainstay of treatment.

Educational objectives: At the end of this practice oral boards case, the learner will:1) recognize unstable vital signs (VS) and intervene to stabilize ventilation and oxygenation, 2) demonstrate the ability to obtain a complete medical history including the important characteristics of chest pain, 3) demonstrate an appropriate exam on a patient, 4) order the appropriate evaluation studies for a patient with complaints of dyspnea, 5) interpret the results of diagnostic evaluation and diagnose Non- ST elevation myocardial infarction (NSTEMI) and pulmonary edema, 6) order appropriate management of pulmonary edema and NSTEMI, and 6) demonstrate effective communication with patient and family members.

Educational methods: Practice oral boards.

Research methods: Immediate Feedback was solicited from the learners and observers participating in the case both by verbal discussion and completion of a rating for the case following the debriefing. The efficacy of the educational content was assessed by comparing scoring measures across residents based on the training year. Scoring measures of the American College of Graduate Medical Education (ACGME) core competencies were performed using a scale from 1 - 8, 1-4 being unacceptable performance and 5 - 8 being acceptable. Efficacy was assumed based on full completion of the case by the residents who acted as practice oral board candidates, and a debriefing session followed to discuss the key components of the case.

Results: This case was presented to twelve Emergency Medicine Residents, seven PGY 1 and five PGY 2 at a relatively new residency program. The overall average score for the residents was 5.62. The PGY 1 Residents' average on the case was 5.56, and the average for the PGY 2 Residents was slightly better at 5.70. The slight improvement noted by the PGY 2 Residents is likely attributable to more clinical experience; however, both classes did not have any prior exposure to the oral board format until this simulated experience. Six residents completed all critical actions in the case. Of those who missed a critical action, failing to diagnose NSTEMI and consulting cardiology were the most common. All learners found educational value in the case with an overall rating of 4.83 (1-5 Likert scale, 5 being excellent).

Discussion: Acute pulmonary edema and NSTEMI are common diagnoses that will be frequently encountered for most emergency physicians. This case highlights the need for early identification and aggressive management of the patient presenting with respiratory distress. The differential for respiratory distress is large, but most learners were able to quickly identify pulmonary edema based on the exam findings of jugular vein distention (JVD), rales, and lower extremity edema. Most learners quickly escalated to a non-rebreather mask and ultimately to BPAP (bilevel positive airway pressure) without requesting to intubate the patient. There was notable variation in the approach to administering nitrates, but most ordered an intravenous (IV) nitroglycerin (NTG) drip and requested pharmacy assistance in dosing. Diuretics were ordered by all the learners, but some were hesitant to start early because they felt the effect would be delayed. Some of the residents did not identify ischemic changes on the EKG at first glance but did request to review a second time when the troponin result was positive. All residents gave aspirin after noting the positive troponin, but not all were able to make a clear diagnosis of NSTEMI or consult cardiology. Although the case was relatively straightforward, residents enjoyed early diagnosis and aggressive management of the patient with impending respiratory failure. Many residents are asking for an ultrasound early in the workup of this patient presenting in respiratory distress. Although not a critical action in this case, it highlights the emphasis placed on ultrasonography in the current emergency medicine curriculum.

Topics: Pulmonary Edema, Cardiovascular emergencies, NSTEMI.

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