远端C1-2层下金属丝固定术后迟发性脊髓病:一例例证。

Aaron Miller, David A Paul, Prasanth Romiyo, Jonathan J Stone
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引用次数: 0

摘要

背景:Atlantaxial亚层布线并发症,包括早期和延迟,已被记录在案。然而,在成功融合27年后,延迟的神经系统损害是罕见但可能发生的。观察结果:一名76岁的男性,1995年因寰枢椎不稳定接受C1-2亚层金属丝融合术,在一周内出现进行性右臂无力、跌倒和大小便失禁的症状。初步影像学检查显示C1-2亚层钢丝弯曲,导致颈脊髓受压和T2加权信号变化。进行C1-2椎板切除术以移除金属丝并对脊髓减压,从而改善患者的神经状态。经验教训:这种罕见的病例突出了即使在成功融合后,层下金属丝也可能导致延迟性颈脊髓病和脊髓压迫。对于有亚层布线史且出现新的神经系统缺陷的患者,评估迁移硬件至关重要。
本文章由计算机程序翻译,如有差异,请以英文原文为准。

Delayed myelopathy after remote C1-2 sublaminar wire fixation: illustrative case.

Delayed myelopathy after remote C1-2 sublaminar wire fixation: illustrative case.

Background: Atlantoaxial sublaminar wiring complications, both early and delayed, have been documented. However, delayed neurological compromise 27 years after successful fusion is a rare but possible occurrence.

Observations: A 76-year-old male, who had undergone C1-2 sublaminar wire fusion for atlantoaxial instability in 1995, presented with symptoms of progressive right arm weakness, falls, and incontinence of bowel and bladder over a 1-week period. Initial imaging workup revealed bowing of the C1-2 sublaminar wires resulting in cervical spinal cord compression and T2-weighted signal changes. A C1-2 laminectomy was performed to remove the wires and decompress the spinal cord with improvement in the patient's neurological status.

Lessons: This rare case highlights the potential for delayed cervical myelopathy and cord compression from sublaminar wires, even after a successful fusion. In patients with a history of sublaminar wiring who experience new neurological deficits, it is essential to evaluate the hardware for migration.

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