肾移植中最低尿酸及文献回顾。

IF 0.3 Q4 TRANSPLANTATION
J Roozbeh, A Dehghani, A Haghpanah, L Malekmakan
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引用次数: 0

摘要

遗传性低尿酸血症是由肾性低尿酸或黄嘌呤尿引起的。黄嘌呤尿分为黄嘌呤脱氢酶缺乏型和黄嘌呤脱氢酶和醛氧化酶缺乏型。我们报告一例黄嘌呤尿1型发展与肾功能衰竭。虽然进行了血液透析,但肾脏功能没有改善,因此进行了肾脏移植手术。移植前后血清尿酸均为0.1 mg/dl。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
The lowest uric acid in kidney transplant and review of literature.

Heredity hypouricemia is caused by renal hypouricemia or xanthinuria. Xanthinuria is divided into type 1 with deficiency of xanthine dehydrogenase and type 2 with xanthine dehydrogenase and aldehyde oxidase deficiency. We report a case of xanthinuria type 1 that developed with kidney failure. Hemodialysis was done for him, but kidney function was not improved, so a kidney transplant was performed for him. His serum uric acid was 0.1 mg/dl before and after transplantation.

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来源期刊
CiteScore
1.60
自引率
0.00%
发文量
0
审稿时长
12 weeks
期刊介绍: The International Journal of Organ Transplantation Medicine (IJOTM) is a quarterly peer-reviewed English-language journal that publishes high-quality basic sciences and clinical research on transplantation. The scope of the journal includes organ and tissue donation, procurement and preservation; surgical techniques, innovations, and novelties in all aspects of transplantation; genomics and immunobiology; immunosuppressive drugs and pharmacology relevant to transplantation; graft survival and prevention of graft dysfunction and failure; clinical trials and population analyses in the field of transplantation; transplant complications; cell and tissue transplantation; infection; post-transplant malignancies; sociological and ethical issues and xenotransplantation.
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