扩张型心肌病患者CTSB启动子多态性与功能的相关性分析

IF 2.6 4区 生物学 Q3 BIOCHEMISTRY & MOLECULAR BIOLOGY
Yu Zhou, Shuang Gao, Liangcai Ding, Han Yan, Shuchao Pang, Bo Yan
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引用次数: 0

摘要

扩张型心肌病(DCM)由遗传易感性和环境因素共同引起。组织蛋白酶B影响DCM的发病机制;然而,其分子机制尚不清楚。在这项研究中,我们研究了罕见的CTSB变异与DCM发生的关系。这项病例对照研究涉及394名参与者:142名DCM患者和252名健康对照者。从所有参与者的外周白细胞中提取DNA,并使用聚合酶链反应扩增分析和鉴定CTSB变异。使用双荧光素酶报告基因法进行功能分析,并使用电泳迁移迁移试验(EMSA)分析和验证遗传CTSB变异与转录因子(TFs)结合的能力。在研究人群中发现了两个单核苷酸多态性(snp)。一个SNP, g.4803T > C (rs1293312)在DCM患者中更为常见。第二个SNP, g.4954在2例DCM患者中鉴定出T > A (rs942670850)。这两个snp都显著增强了CTSB启动子的转录活性。使用TRANSFAC数据库进行的分析显示,这些snp影响TF结合,这一点通过EMSA得到了证实。我们的研究结果表明,在CTSB启动子内,遗传变异g.4803T>C (rs1293312)和g.4954T > A (rs942670850)是DCM发展的罕见危险因素。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Correlation Analysis of CTSB Promoter Polymorphism and Function in Patients with Dilated Cardiomyopathy.

Dilated cardiomyopathy (DCM) is caused by a combination of genetic susceptibility and environmental factors. Cathepsin B affects the pathogenesis of DCM; however, its molecular mechanism is still unclear. In this study, we examined the association of rare CTSB variants with the occurrence of DCM. This case-control study involved 394 participants: 142 patients with DCM and 252 healthy controls. DNA was extracted from the peripheral leukocytes of all participants, and CTSB variants were analyzed and identified using polymerase chain reaction amplification. Functional analysis was performed using the dual-luciferase reporter assay, and the ability of genetic CTSB variants to bind to transcription factors (TFs) was analyzed and validated using the electrophoretic mobility shift assay (EMSA). Two single-nucleotide polymorphisms (SNPs) were identified in the study population. One SNP, g.4803 T > C (rs1293312), was more common in patients with DCM. A second SNP, g.4954 T > A (rs942670850), was identified in two patients with DCM. Both SNPs significantly enhanced the transcriptional activity of CTSB promoters. An analysis using the TRANSFAC database revealed that these SNPs affect TF binding, which was confirmed using the EMSA. Our results demonstrate that within the CTSB promoter, the genetic variants g.4803T>C (rs1293312) and g.4954 T > A (rs942670850) are rare risk factors for DCM development.

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来源期刊
DNA and cell biology
DNA and cell biology 生物-生化与分子生物学
CiteScore
6.60
自引率
0.00%
发文量
93
审稿时长
1.5 months
期刊介绍: DNA and Cell Biology delivers authoritative, peer-reviewed research on all aspects of molecular and cellular biology, with a unique focus on combining mechanistic and clinical studies to drive the field forward. DNA and Cell Biology coverage includes: Gene Structure, Function, and Regulation Gene regulation Molecular mechanisms of cell activation Mechanisms of transcriptional, translational, or epigenetic control of gene expression Molecular Medicine Molecular pathogenesis Genetic approaches to cancer and autoimmune diseases Translational studies in cell and molecular biology Cellular Organelles Autophagy Apoptosis P bodies Peroxisosomes Protein Biosynthesis and Degradation Regulation of protein synthesis Post-translational modifications Control of degradation Cell-Autonomous Inflammation and Host Cell Response to Infection Responses to cytokines and other physiological mediators Evasive pathways of pathogens.
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