Covid-19后女性并发症

Ajay I. Patel, Anju Yadav, Ashok B. Patel, Amit J. Vyas
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引用次数: 0

摘要

COVID-19后是指急性COVID-19后持续存在或恶化的临床症状,包括持续4至12周或COVID-19后超过12周的症状性COVID-19。截至2020年6月,鉴于“后COVID-19”这一术语。已报道的研究与女性患者的并发症有关,包括对免疫系统、生理或心理健康的影响,以及对已有疾病的影响,包括高血压、糖尿病、神经退行性疾病、类风湿关节炎和结核病(TB)。然而,在COVID-19康复六个月后,出现并发症,包括咳嗽、发烧、呼吸困难、肌肉疼痛、关节疼痛、疲劳、胃肠道不适、嗅觉减退、消化不良、睡眠困难、焦虑或抑郁、压力水平升高、身体衰退、活动后呼吸急促、脱发、胸闷、心悸、肠梗阻、呼吸功能受损、神经问题、嗅觉功能障碍、认知、灵活性、对话、视力障碍、或倾听。还有其他各种并发症,比如米勒-费雪综合征。总的来说,这些综述总结了COVID-19后女性患者的既往疾病、女性免疫功能障碍患者的并发症(包括激素失衡)以及女性神经退行性并发症患者的csf不足的研究。观察到高水平的分子标记,如tau蛋白(t-tau, p-tau),血浆淀粉样蛋白(A42),胶质纤维酸性蛋白(GFAP),泛素c端水解酶- l1 (UCH-L1),神经丝轻链蛋白。合并糖尿病(1型和2型)并发症的女性患者中,sCr、c反应蛋白、tni、白细胞、红细胞沉降率、酶、电解质和凝血因子水平较高,合并结核病(TB)并发症的女性患者侵袭性血管粘液瘤(AAM)和E2激素水平较低。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Post Covid-19 Women Complications
Post-covid-19 women complications, Post covid-19 is Clinical symptoms that persist or worsen after acute COVID It is contains both ongoing symptomatic COVID-19 between 4 to 12 weeks or post COVID-19 more than 12 weeks. As of June 2020, given the terminology "post COVID-19". Reported studies are related to complications of the female patient, including effects on the immune system, physiology, or psychological health, and effects on pre-existing diseases, including hypertension, diabetes mellitus, neurodegenerative disease, rheumatoid arthritis, and tuberculosis (TB). However, six months after recovery from COVID-19, complications arise, including cough, fever, breathlessness, muscle aches, joint pain, fatigue, gastrointestinal complaints, anosmia, dyspepsia, sleep difficulties, anxiety or depression, higher stress levels, physical decline, post-activity polypnea, alopecia, chest tightness, palpitations, intestinal blockages, impaired respiratory functions, neurological issues, olfactory dysfunction, cognition, dexterity, conversation, disabilities of sight, or listening. Along with other miscellaneous complications such as Miller-Fisher syndrome. Overall, these reviews summarise studies conducted on pre-existing diseases in female patients after COVID-19, complications in female patients with immune dysfunction, including hormonal imbalances, and CSF-insufficiency in female patients with neurodegeneration complications. It observed high levels of molecular markers such as tau protein (t-tau, p-tau), plasma amyloid-beta (A42), glial fibrillary acidic protein (GFAP), ubiquitin C-terminal hydrolase-L1 (UCH-L1), neurofilament light chain protein. In Female patients with diabetes mellitus (Type 1 and Type 2) complications reported higher levels of sCr, C-reactive protein, TN-I, white blood cell, and erythrocyte sedimentation rate levels, enzymes, electrolytes, and coagulation factors, and female patients with tuberculosis (TB) complications had lower aggressive angiomyxoma (AAM) and E2 hormone levels.
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