心理健康、心血管风险因素与大学生

H. H. Betz, V. Leadbetter, J. Cousins
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引用次数: 0

摘要

社交体质焦虑(SPA)与身体活动(PA)行为和焦虑障碍症状相关。然而,关于SPA对PA和广泛性焦虑障碍(GAD)之间关联的潜在影响知之甚少。目的:本研究量化了年轻人PA、GAD和SPA之间的关系(N=470, 23.2±4.8y;63.4%女性),并探讨SPA作为PA和GAD之间关联的中介。方法:7天PA召回确定估计支出(千卡/周),并分类非活性、中等活性和高度活性PA剂量类别。精神病学诊断筛查问卷GAD亚量表评估GAD症状严重程度;得分≥6表示类似GAD (AGAD)状态。社会体质焦虑量表评估SPA。独立t检验检验了基于性别和AGAD状态的基线差异。Cohen 's d量化了差异的大小。Logistic回归根据PA剂量量化AGAD的几率,调整年龄、性别和吸烟状况。简单中介分析考察了SPA对PA-GAD症状持续关联的中介作用。结果:女性报告的PA (p≤0.002,d=0.31)和SPA (p≤0.001,d=-0.63)和GAD症状严重程度(p≤0.001,d=-0.51)较低。AGAD报告更高的SPA (p≤0.001,d=0.92)。与不运动组相比,中度运动组和高度运动组AGAD的发生率分别降低28.3% (OR=0.72, 95%CI: [0.43, 1.20], p≥0.21)和42.5% (OR=0.58, [0.35, 0.94], p≤0.03)。在调整后的模型中,与不运动患者相比,中度运动和高度运动患者的比值分别为29.3% (OR= 0.71, [0.42, 1.20], p≥0.21)和36.9% (OR=0.63, [0.38, 1.06], p≥0.08)。PA对GAD症状的回归模型(β=-0.01, p≤0.04)、SPA对PA (β=-0.02, p≤0.03)、SPA对GAD症状的回归模型(β=0.14, p≤0.001)均有显著性意义。共同回归时,SPA为(β=0.14, p≤0.001),PA无(β=-0.003, p≤0.27),具有统计学意义,支持中介作用。结论:PA可能降低GAD的发生率,但在调整协变量后发现不显著。SPA是一种可改变因子,在女性和AGAD患者中较高,介导了PA和GAD之间的关联。未来的研究应纵向考察这些关系,并通过实验探索SPA。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Mental Health, Cardiovascular Risk Factors, And The College Student
Social Physique Anxiety (SPA) has been associated with physical activity (PA) behaviors and anxiety disorder symptoms. However, little is known about the potential influence of SPA on associations between PA and Generalized Anxiety Disorder (GAD). PURPOSE: This study quantified associations between PA, GAD and SPA among young adults (N=470, 23.2±4.8y; 63.4% female) and explored SPA as a mediator of the association between PA and GAD. METHODS: Seven-day PA Recall determined estimated expenditure (kcal/wk) and classified inactive, moderately active, and highly active PA dose categories. The Psychiatric Diagnostic Screening Questionnaire GAD subscale assessed GAD symptom severity; a score of ≥6 indicated analogue GAD (AGAD) status. The Social Physique Anxiety Scale assessed SPA. Independent t-tests examined baseline differences based on gender and AGAD status. Cohen‘s d quantified the magnitude of difference. Logistic regression quantified odds of AGAD based on PA dose, adjusting for age, gender, and smoking status. Simple mediation analyses examined mediation of the continuous PA-GAD symptom association by SPA. RESULTS: Females reported less PA (p≤0.002, d=0.31) and greater SPA (p≤0.001, d=-0.63) and GAD symptom severity (p≤0.001, d=-0.51). AGAD reported greater SPA (p≤0.001, d=0.92). Compared to inactive, odds of AGAD were 28.3% (OR=0.72, 95%CI: [0.43, 1.20], p≥0.21) and 42.5% (OR=0.58, [0.35, 0.94], p≤0.03) lower among moderately active and highly active, respectively. In adjusted models, compared to inactive, odds were 29.3% (OR= 0.71, [0.42, 1.20], p≥0.21) and 36.9% (OR=0.63, [0.38, 1.06], p≥0.08) lower among moderately active and highly active, respectively. Regression models of PA on GAD symptoms (β=-0.01, p≤0.04), SPA on PA (β=-0.02, p≤0.03), and SPA on GAD symptoms (β=0.14, p≤0.001) were significant. When regressed together, SPA was (β=0.14, p≤0.001), but PA was not (β=-0.003, p≤0.27), statistically significant, supporting mediation. CONCLUSION: PA may lower odds of GAD, but findings were not significant after adjusting for covariates. SPA, a modifiable factor that was higher among females and those with AGAD, mediated the association between PA and GAD. Future research should examine these relationships longitudinally and explore SPA experimentally.
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