“及时识别可改变的高钾血症”-受体阻滞剂(琥珀酸美托洛尔)诱导

P. Prasad, S. Raj
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引用次数: 0

摘要

本病例报告描述了非常罕见的药物诱导高钾血症与受体阻滞剂的可能性。在文献检索中,仅有2例无肾功能不全的糖尿病患者被报道为琥珀酸美托洛尔所致高钾血症。根据FDA的报告,只有0.5%的患者报告美托洛尔引起的高钾血症。患者有糖尿病和高血压。她正在服用口服降糖药,预混胰岛素和受体阻滞剂以及钙通道阻滞剂。钾值为6 ~ 6.7 mmol/l。详细评估,实验室检查未发现其他异常。一旦停用药物美托洛尔,钾值在很短时间内恢复到正常范围。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
“Modifiable Hyperkalemia with Timely Recognition”- Beta Blocker (Metoprolol succinate) induced
This case report describes the very rarest possibility of drug induced hyperkalemia with beta blockers. In literature search only 2 cases were reported as metoprolol succinate induced hyperkalemia in diabetes patients without renal insufficiency. As per FDA reports  only 0.5 % patients have reported hyperkalemia induced by metoprolol [1]. Patient is diabetic and hypertensive. And she is on oral hypoglycemic agents, premixed insulin and on beta blockers along with calcium channel blocker. The potassium value was ranging from 6 to 6.7 mmol/l. Evaluated in detail and no other abnormalities noted in laboratory investigations. Once the offending drug metoprolol is withdrawn the potassium value reaches to normal range in very short period.
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