SARS-CoV-2刺突蛋白循环技术的心脏转移-药物诱导血液透析对“接种”免疫攻击的影响

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引用次数: 1

摘要

背景:报告1例SARS-CoV-2疫苗接种病例。由于sars冠状病毒的Spike 2 (S2)蛋白与HIV-1 gp41具有相似的螺旋结构,患者在第三次接种科星重组疫苗一年后出现了严重的免疫系统反应。方法:根据患者的症状构思药物诱导血液透析技术,经紧急治疗后实施。患者紧急治疗的指标证实了初始处方的有效性。结果:临床病例验证了药物诱导血液透析方法治疗SARS-CoV(-2)相关早期症状在减少对人体不可逆损害方面的作用。结论:临床病例和文献复习提示SARS-CoV系列是不可能来自自然来源的复合病毒。据推测,该病毒至少可以追溯到上世纪90年代在中国大陆传播,可能是核试验的结果。对SARS-CoV-2的S2蛋白和流行病学品种的低估可能导致全球疫苗需求,对人类免疫系统的损害需要进一步研究。研究认为,sars病毒证明了中国违反了1948年《日内瓦公约》,这是中共政权及其执政党系统地、故意地对社会和国际机构撒谎的原因。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Cardiac Transfer of SARS-CoV-2 Spike Protein Circulation Techniques — Medicine Induced Hemodialysis on “Vaccinated” Immune Attacks
Background: The research reports on a medical case of SARS-CoV-2 vaccinated patient. With the SARS-CoV’s Spike 2 (S2) protein sharing similar helix structure with HIV-1 gp41, the patient experienced severe immune system responses one year after the third dose of Kexing recombined vaccines. Methods: The author conceived a medicine induced hemodialysis technique according to the patient’s symptoms, and implemented after emergent treatments. Indicators from the patient’s emergent treatments corroborate with the initial prescription’s validity. Results: The clinical case validates the medicine induced hemodialysis method in treating SARS-CoV(-2)-related early symptoms in reducing irreversible damages to humans, depending on individual clinical cases. Conclusions: The clinical case along with literature review suggests SARS-CoV series are compound virus unlikely from natural origins. It is inferred to have spread in mainland China at least back to the 1990s, and possibly a result from nuclear tests. The downplay of SARS-CoV-2’s S2 protein and epidemiological varieties could have led to the global vaccine mandates, and the damages to human immune system need to be further researched into. The research concludes that SARS-CoV evidenced PRC’s transgressions to the 1948 Geneva Conventions, which is the reason that the regime and its ruling party are systematically and willfully lying to the society and global institutions.
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