喉主动脉压力感受器通路与心律失常。

J. Jacobs, A. Wetzel, M. Hast
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引用次数: 4

摘要

本研究旨在证实交通支压力感受器通路在上呼吸道障碍时对心率的影响。我们测试了破坏这条通路的效果。结果发现,尽管没有交通支纤维,但刺激喉上神经主干仍能改变心率。电刺激喉上神经的主干,而不是它的任何分支,能够引起心脏的变化。尽管双侧喉返神经和喉上神经被破坏,但对声带上方区域的机械刺激也容易发生心脏变化。这些发现质疑喉部压力感受器通路作为插管引起心脏改变的主要因素的作用。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Laryngeal aortic baroreceptor pathways and cardiac arrhythmia.
This study was undertaken to confirm the effect of the ramus communicans baroreceptor pathway on cardiac rate during an upper-respiratory tract disturbance. The effects of destruction of this pathway were tested. It was found that cardiac rate could still be altered by stimulation of the main trunk of the superior laryngeal nerve, although the ramus communicans fibers were absent. Electrical stimulation of only the main trunk of the superior laryngeal nerve, but not any of its branches, is capable of causing cardiac changes. Cardiac changes also occur readily with mechanical stimulation of the area immediately superior to the vocal folds, despite bilateral destruction of the recurrent and superior laryngeal nerves. These findings question the proposed role of the laryngeal baroreceptor pathway as a major factor in the cause of cardiac changes related to intubation.
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