{"title":"心外膜脂肪组织与心房颤动的关系","authors":"I. Abe, S. Miyamoto, N. Takahashi","doi":"10.5105/jse.40.149","DOIUrl":null,"url":null,"abstract":"Several lines of evidence have shown that epicardial adipose tissue（EAT）is related to the presence, severity, and outcome of atrial fibrillation（AF）. Mechanistically, studies have suggested that the effects of EAT may be mediated by local cytokines/chemokines, inflammation, and fatty infiltration. Here, we discuss the role of EAT, especially in its effect on atrial myocardial fibrosis. Prevention of qualitative alteration of EAT may be a novel therapeutic target for AF.","PeriodicalId":14543,"journal":{"name":"Japanese Journal of Electrocardiology","volume":"170 1","pages":""},"PeriodicalIF":0.0000,"publicationDate":"2020-10-30","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":"{\"title\":\"Association Between Epicardial Adipose Tissue and Atrial Fibrillation\",\"authors\":\"I. Abe, S. Miyamoto, N. Takahashi\",\"doi\":\"10.5105/jse.40.149\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"Several lines of evidence have shown that epicardial adipose tissue（EAT）is related to the presence, severity, and outcome of atrial fibrillation（AF）. Mechanistically, studies have suggested that the effects of EAT may be mediated by local cytokines/chemokines, inflammation, and fatty infiltration. Here, we discuss the role of EAT, especially in its effect on atrial myocardial fibrosis. Prevention of qualitative alteration of EAT may be a novel therapeutic target for AF.\",\"PeriodicalId\":14543,\"journal\":{\"name\":\"Japanese Journal of Electrocardiology\",\"volume\":\"170 1\",\"pages\":\"\"},\"PeriodicalIF\":0.0000,\"publicationDate\":\"2020-10-30\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Japanese Journal of Electrocardiology\",\"FirstCategoryId\":\"1085\",\"ListUrlMain\":\"https://doi.org/10.5105/jse.40.149\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"\",\"JCRName\":\"\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Japanese Journal of Electrocardiology","FirstCategoryId":"1085","ListUrlMain":"https://doi.org/10.5105/jse.40.149","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
Association Between Epicardial Adipose Tissue and Atrial Fibrillation
Several lines of evidence have shown that epicardial adipose tissue（EAT）is related to the presence, severity, and outcome of atrial fibrillation（AF）. Mechanistically, studies have suggested that the effects of EAT may be mediated by local cytokines/chemokines, inflammation, and fatty infiltration. Here, we discuss the role of EAT, especially in its effect on atrial myocardial fibrosis. Prevention of qualitative alteration of EAT may be a novel therapeutic target for AF.
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