{"title":"眼麻痹性偏头痛和青少年肌阵挛性癫痫:同一枚硬币的两面?","authors":"A. Deshpande, Supriya Khardenavis, Aakash Shetty","doi":"10.14800/MCE.70","DOIUrl":null,"url":null,"abstract":"Excessive neocortical cellular excitability secondary to complex molecular mechanisms involving Sodium Pottasium ATPase & elevated glutamate ,potassium levels in extracellular fluid form the possible common underlying mechanism for causation of ophthalmoplegic migraine & juvenile myoclonic epilepsy both alike and further research is needed to establish these hypothesis in the future.","PeriodicalId":18603,"journal":{"name":"Molecular & Cellular Epilepsy","volume":"31 1","pages":""},"PeriodicalIF":0.0000,"publicationDate":"2014-03-05","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":"{\"title\":\"Ophthalmoplegic Migraine and Juvenile Myoclonic Epilepsy: Two side of the same coin?\",\"authors\":\"A. Deshpande, Supriya Khardenavis, Aakash Shetty\",\"doi\":\"10.14800/MCE.70\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"Excessive neocortical cellular excitability secondary to complex molecular mechanisms involving Sodium Pottasium ATPase & elevated glutamate ,potassium levels in extracellular fluid form the possible common underlying mechanism for causation of ophthalmoplegic migraine & juvenile myoclonic epilepsy both alike and further research is needed to establish these hypothesis in the future.\",\"PeriodicalId\":18603,\"journal\":{\"name\":\"Molecular & Cellular Epilepsy\",\"volume\":\"31 1\",\"pages\":\"\"},\"PeriodicalIF\":0.0000,\"publicationDate\":\"2014-03-05\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Molecular & Cellular Epilepsy\",\"FirstCategoryId\":\"1085\",\"ListUrlMain\":\"https://doi.org/10.14800/MCE.70\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"\",\"JCRName\":\"\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Molecular & Cellular Epilepsy","FirstCategoryId":"1085","ListUrlMain":"https://doi.org/10.14800/MCE.70","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
Ophthalmoplegic Migraine and Juvenile Myoclonic Epilepsy: Two side of the same coin?
Excessive neocortical cellular excitability secondary to complex molecular mechanisms involving Sodium Pottasium ATPase & elevated glutamate ,potassium levels in extracellular fluid form the possible common underlying mechanism for causation of ophthalmoplegic migraine & juvenile myoclonic epilepsy both alike and further research is needed to establish these hypothesis in the future.