鉴定与马凡氏综合征相关的 FBN1 新型致病变体。

IF 1.8 Q3 MEDICINE, RESEARCH & EXPERIMENTAL
Julia P Pereira, Juliana R Ferreira, Anna Paula A Botelho, Marcelo M Melo, Glauber Monteiro Dias
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引用次数: 0

摘要

马凡综合征(MFS)引起的主动脉疾病,如胸主动脉瘤和主动脉夹层,与 FBN1 基因的遗传改变有关。通用突变-FBN1、ClinVar 和人类基因突变等数据库中包含了一千多个与 MFS 相关的 FBN1 基因突变。FBN1 基因编码纤维蛋白-1,负责不同蛋白质结构域的整体生成。可能的基因变化会导致血管变弱,从而引发主动脉病变。在这项研究中,我们发现了一种新型 FBN1 变异与 MFS 的关联。原发者是一名男性,42 岁时出现升主动脉瘤和夹层(TAAD),后经手术治疗。所有参与研究的家庭成员都接受了临床检查。在原告、女儿和孙女身上都发现了马凡氏征。对该患者的 FBN1 基因进行了直接测序,发现了一个新的截断变异 p.(Glu2019Ter),并进行了级联筛选。根据美国医学遗传学和基因组学学会(ACMG)的标准和修订后的根特 MFS 诊断命名法,该变异体分别被归类为 MFS 的致病变异体和因果变异体。普罗班德的女儿和孙女也携带这种变异,但没有主动脉改变。本研究首次报道了一名 FBN1-p.(Glu2019Ter) 变异患者及其与 MFS/TAAD 的关系。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Identification of a novel pathogenic variant in FBN1 associated with Marfan Syndrome.

Aortic diseases arising in Marfan Syndrome (MFS), such as in aneurysms and dissections of the thoracic aorta, are related to genetic alterations in the FBN1 gene. Databases, such as Universal Mutations-FBN1, ClinVar and The Human Gene Mutation, contain more than a thousand FBN1 mutations associated with MFS. The FBN1 gene, which encodes fibrillin-1, is responsible for the integral production of different protein domains. Possible genetic changes may lead to a weakening of blood vessels, leading to the development of aortopathies. In this study, we present the association of a novel FBN1 variant with MFS. The proband is a man who presented ascending aortic aneurysm and dissection (TAAD) at 42-yr-old, which was surgically treated. Clinical investigations were performed in all family members enrolled in the study. Marfan signs were observed in the proband, daughters and granddaughter. Direct sequencing of the FBN1 gene in the proband identified a novel truncation variant p.(Glu2019Ter) and a cascade screening were done. The variant was classified as pathogenic and causal for MFS according to the American College of Medical Genetics and Genomics (ACMG) criteria and revised Ghent nosology for MFS diagnosis, respectively. Proband's daughter and granddaughter harbor the variant, however without aortic alteration. This work reports for the first time a patient with the FBN1-p.(Glu2019Ter) variant and its association with MFS/TAAD.

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来源期刊
Cold Spring Harbor Molecular Case Studies
Cold Spring Harbor Molecular Case Studies MEDICINE, RESEARCH & EXPERIMENTAL-
CiteScore
3.20
自引率
0.00%
发文量
54
期刊介绍: Cold Spring Harbor Molecular Case Studies is an open-access, peer-reviewed, international journal in the field of precision medicine. Articles in the journal present genomic and molecular analyses of individuals or cohorts alongside their clinical presentations and phenotypic information. The journal''s purpose is to rapidly share insights into disease development and treatment gained by application of genomics, proteomics, metabolomics, biomarker analysis, and other approaches. The journal covers the fields of cancer, complex diseases, monogenic disorders, neurological conditions, orphan diseases, infectious disease, gene therapy, and pharmacogenomics. It has a rapid peer-review process that is based on technical evaluation of the analyses performed, not the novelty of findings, and offers a swift, clear path to publication. The journal publishes: Research Reports presenting detailed case studies of individuals and small cohorts, Research Articles describing more extensive work using larger cohorts and/or functional analyses, Rapid Communications presenting the discovery of a novel variant and/or novel phenotype associated with a known disease gene, Rapid Cancer Communications presenting the discovery of a novel variant or combination of variants in a cancer type, Variant Discrepancy Resolution describing efforts to resolve differences or update variant interpretations in ClinVar through case-level data sharing, Follow-up Reports linked to previous observations, Plus Review Articles, Editorials, and Position Statements on best practices for research in precision medicine.
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