6岁儿童盐中毒1例报告

A. Alqahtani, M. Molla, Faisal M Alshiban
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摘要

背景:盐中毒引起的高钠血症在临床上很少见,标准的治疗程序尚未建立。我们报告这个儿童盐中毒的情况下,由于长期渴望食盐。立即认识到盐的毒性并采取积极的复苏措施是治疗这种致命中毒的关键。尽管采取了英勇的措施,但由于盐毒性导致的儿童死亡仍然发生在不可逆转的神经损伤中。病例介绍:一名6岁男童从外院转诊至我儿科急诊科,因疲劳、多尿、烦渴及明显的高钠血症,血清钠浓度203 mmol/L。最初的实验室检查显示血糖5.7 mmol/l,血清钠(203 mmol/l)来自转诊医院)和静脉气显示PH 7.26 PCO2 47 mmHg HCO3 21.5 mmol/l。我们在84小时内缓慢降低了他的血清钠水平,因为它的毒性持续了2个月。结论:在回顾了盐中毒后的复苏实例后,只有在摄入盐中毒后几小时内的高钠血症急性期才应考虑积极快速纠正血清钠浓度。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Salt Poisoning in a 6-year-old Child: A Case Report
Background: Hypernatremia due to salt poisoning is clinically rare and standard care procedures have not been established. We report this case of pediatric salt poisoning due to chronic craving for table salt. Immediate recognition of salt toxicity and aggressive resuscitative measures are critical in the treatment of this lethal poisoning. Despite heroic measures, pediatric deaths due to salt toxicity still occur from irreversible neurological damage. Case presentation: A 6-year-old boy presented to our pediatric emergency department referred from other hospital with fatigue, polyuria and polydipsia and remarkable hypernatremia with a serum sodium concentration of 203 mmol/L. The initial lab work showed blood glucose 5.7 mmol/l, Serum Sodium (203 mmol/l) from referring hospital) and venous blood gases showed PH 7.26 PCO2 47 mmHg HCO3 21.5 mmol/l. We reduced his serum sodium level slowly over 84 hours, as its toxicity happened over a period of 2 months. Conclusion: After reviewing instances of resuscitation following salt intoxication, aggressive rapid correction of serum sodium concentration should only be considered in acute phases of hypernatremia within a few hours from ingestion.
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