Sars-Cov2尖峰和端粒酶rnaandsquo的比较得出了严重COVID-19患者肺泡细胞老化加剧的解释

H. Geurdes
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引用次数: 0

摘要

在这封信中,我们研究了SARS-CoV-2 RNA是否参与肺泡细胞的加速老化。我们的计算机研究是探索性的。结果表明,AEC2对博来霉素损伤肺泡细胞的修复具有重要意义。如果AEC2修复能力被刺突RNA削弱,那么这一结果可能为治疗不死肺癌细胞提供了第一步。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Sars-Cov2 Spike and Telomerase RNAandrsquo;s Compared to Arrive at an Explanation for Increased Ageing in Alveolar Cells in Severe COVID-19
In this letter we investigate if SARS-CoV-2 RNA is involved in the increased ageing of alveolar cells. Our in silico study is explorative. With the results we are able to outline experiments with AEC2 repair of bleomycin damaged alveolar cells. If AEC2 repair capability is diminished by spike RNA then perhaps this result provides a first step on a route to treat immortal lung cancer cells.
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