ninninyoeito通过神经生长因子减少β -淀粉样蛋白25 - 35诱导的轴突损伤

Kaori Kubota, Takuya Watanabe, S. Katsurabayashi, K. Iwasaki
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引用次数: 1

摘要

日本的草药“NYT”被广泛用于治疗失眠、贫血、健忘症、神经症等疾病。最近,NYT被报道对阿尔茨海默病(AD)有临床疗效。我们研究了NYT在神经变性模型中的作用,该模型包括暴露于β -淀粉样蛋白(a β) 25-35的培养自噬神经元。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Ninjinyoeito reduces β‐amyloid25–35‐induced axon damage via nerve growth factor
Ninjinyoeito (NYT), a Japanese herbal medicine, is widely used to treat patients with insomnia, anemia, amnesia, and neurosis. Recently, NYT was reported to be clinically effective in Alzheimer's disease (AD). We investigated the effects of NYT in a neurodegeneration model comprising cultured autaptic neurons exposed to β‐amyloid (Aβ)25–35.
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