{"title":"肝炎病毒序列分析","authors":"J. Varela","doi":"10.35248/1948-5964.20.S8.E002","DOIUrl":null,"url":null,"abstract":"Hepatitis B Virus (HBV) could also be a noncytopathic DNA virus that infects approximately 350 million people worldwide and should be a number one explanation for liver cirrhosis and hepatoma. The evolutionary dynamics of HBV are characterized by high mutation rates due to the error-prone polymerase (RT) and rapid replication rates. Thus, an outsized number of viral variants are constantly generated, creating great genetic diversity on which survival operates.","PeriodicalId":15020,"journal":{"name":"Journal of Antivirals & Antiretrovirals","volume":"103 1","pages":"1-1"},"PeriodicalIF":0.0000,"publicationDate":"2020-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":"{\"title\":\"Sequencing Analysis of Hepatitis\",\"authors\":\"J. Varela\",\"doi\":\"10.35248/1948-5964.20.S8.E002\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"Hepatitis B Virus (HBV) could also be a noncytopathic DNA virus that infects approximately 350 million people worldwide and should be a number one explanation for liver cirrhosis and hepatoma. The evolutionary dynamics of HBV are characterized by high mutation rates due to the error-prone polymerase (RT) and rapid replication rates. Thus, an outsized number of viral variants are constantly generated, creating great genetic diversity on which survival operates.\",\"PeriodicalId\":15020,\"journal\":{\"name\":\"Journal of Antivirals & Antiretrovirals\",\"volume\":\"103 1\",\"pages\":\"1-1\"},\"PeriodicalIF\":0.0000,\"publicationDate\":\"2020-01-01\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Journal of Antivirals & Antiretrovirals\",\"FirstCategoryId\":\"1085\",\"ListUrlMain\":\"https://doi.org/10.35248/1948-5964.20.S8.E002\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"\",\"JCRName\":\"\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Journal of Antivirals & Antiretrovirals","FirstCategoryId":"1085","ListUrlMain":"https://doi.org/10.35248/1948-5964.20.S8.E002","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
Hepatitis B Virus (HBV) could also be a noncytopathic DNA virus that infects approximately 350 million people worldwide and should be a number one explanation for liver cirrhosis and hepatoma. The evolutionary dynamics of HBV are characterized by high mutation rates due to the error-prone polymerase (RT) and rapid replication rates. Thus, an outsized number of viral variants are constantly generated, creating great genetic diversity on which survival operates.
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