工业化家禽养殖中鸟类应激的实验模型研究及预防

T. Pogrebnyak, E. Khorolskaya, S. Chernyavskikh, I. Sagalaeva
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引用次数: 0

摘要

在鸟类中,急性应激(三天12小时的光模倒置)与交感下丘脑和海马体的主要活动有关,这抑制了副交感神经部分的张力和中脑的网状形成。在负变时效应的背景下高血糖的影响表明在第30天下丘脑自主神经部分的功能不匹配。鸟类慢性应激(30天的拥挤)导致下丘脑前部功能和中脑网状结构的持续病理应激,下丘脑后部的pap交感神经张力受到抑制,海马的高功能活动,决定了心脏心室在第30天转向更经济的功能模式。排除了其在神经源性应激和应激后时期的影响机制中具有胆碱阻断成分,但可能使用应激保护因子。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Experimental Models of Study and Prevention of Stress in Birds in Industrial Poultry Farming
—In birds, acute stress (a three-day 12-hour inversion of the photomode) is associated with the dominant activity of the sympathetic hypothalamus and hippocampus, which suppress the tone of the parasympathetic section and the reticular formation of the midbrain. The effect of hyperglycemia on the background of a negative chronotropic effect indicates a mismatch of the functions of the autonomic sections of the hypothalamus on the 30 th day. Chronic stress in birds (30-days crowding) causes persistent pathological stress of the functions of the anterior hypothalamus and reticular formation of the middle brain with the suppression of papasympathetic tone of the posterior hypothalamus and high functional activity of the hippocampus, determining the switching of the ventricles of the heart to a more economical mode of functioning by the 30th day. having a cholinoblocking component in the mechanism of its influence during neurogenic stress and in the post-stress period is excluded, but it is possible to use stressprotektors.
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