半月板退行性和水平撕裂修复的可能性

T. Mine, Takaaki Ueda, K. Ihara, H. Kawamura, Ryutaro Kuriyama, Yasunari Tominaga
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引用次数: 1

摘要

由于创伤,半月板被切除后不能再生;继发性骨关节炎的发生是由于半月板功能的丧失,包括负荷分布、冲击吸收、关节滑动和稳定的改变。为了保护半月板,许多外科医生在可能的情况下对红红撕裂和红白撕裂进行半月板修复。然而,半月板退行性撕裂和白色-白色撕裂的修复是具有挑战性的[13]。先前我们报道,在创伤性半月板破裂碎片中,II型和III型胶原蛋白首先消失,其次是I型胶原蛋白,导致纤维结构消失。据推测,这些功能的丧失是因为破裂碎片中半月板细胞的营养供应不足。由于大部分半月板细胞消失,断片的半月板功能不能维持;
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Possibility of Meniscal Repair for Degenerative and Horizontal Tears
The meniscus does not regenerate once it has been removed because of trauma; secondary osteoarthritic changes occur due to the loss of meniscal function, including alterations in load distribution, impact absorption, and articular sliding and stabilization. To preserve the meniscus, many surgeons perform meniscal repairs for red-red tears and red-white tears when possible. However, meniscal repair for degenerative tears and white-white tears is challenging [1 3]. Previously, we reported that in the ruptured fragments of trauma-injured menisci, collagen types II and III disappear first, followed by collagen type I, resulting in the abrogation of fiber construction. It is assumed that these functions are lost because insufficient nutrition is supplied to the meniscal cells in the ruptured fragments. Meniscal function of the rupture fragment cannot be maintained as most meniscal cells have disappeared;
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