{"title":"婴儿长链3-羟基酰基辅酶A脱氢酶缺乏过程中扩张型心肌病后肥厚型心肌病的恢复","authors":"J. Kwiatkowska, Julita Świtońska, J. Wierzba","doi":"10.5430/JBGC.V3N3P26","DOIUrl":null,"url":null,"abstract":"Dilated cardiomyopathy is one of the most common causes of the heart failure in childhood and can develop as a consequence of metabolic disorders such as fatty acid beta-oxidation disorders or amino acids metabolism disorders. In our report we present a case where in the course of deficiency of long-chain 3-hydroxyacyl-coenzyme A dehydrogenase the recovery from dilated cardiomyopathy resulted in hypertrophic cardiomyopathy.","PeriodicalId":89580,"journal":{"name":"Journal of biomedical graphics and computing","volume":"3 1","pages":"26"},"PeriodicalIF":0.0000,"publicationDate":"2013-04-07","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.5430/JBGC.V3N3P26","citationCount":"1","resultStr":"{\"title\":\"The recovery from dilated cardiomyopathy followed by hypertrophic cardiomyopathy, in the course of deficiency of long-chain 3-hydroxyacyl- coenzyme A dehydrogenase in infant\",\"authors\":\"J. Kwiatkowska, Julita Świtońska, J. Wierzba\",\"doi\":\"10.5430/JBGC.V3N3P26\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"Dilated cardiomyopathy is one of the most common causes of the heart failure in childhood and can develop as a consequence of metabolic disorders such as fatty acid beta-oxidation disorders or amino acids metabolism disorders. In our report we present a case where in the course of deficiency of long-chain 3-hydroxyacyl-coenzyme A dehydrogenase the recovery from dilated cardiomyopathy resulted in hypertrophic cardiomyopathy.\",\"PeriodicalId\":89580,\"journal\":{\"name\":\"Journal of biomedical graphics and computing\",\"volume\":\"3 1\",\"pages\":\"26\"},\"PeriodicalIF\":0.0000,\"publicationDate\":\"2013-04-07\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"https://sci-hub-pdf.com/10.5430/JBGC.V3N3P26\",\"citationCount\":\"1\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Journal of biomedical graphics and computing\",\"FirstCategoryId\":\"1085\",\"ListUrlMain\":\"https://doi.org/10.5430/JBGC.V3N3P26\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"\",\"JCRName\":\"\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Journal of biomedical graphics and computing","FirstCategoryId":"1085","ListUrlMain":"https://doi.org/10.5430/JBGC.V3N3P26","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
The recovery from dilated cardiomyopathy followed by hypertrophic cardiomyopathy, in the course of deficiency of long-chain 3-hydroxyacyl- coenzyme A dehydrogenase in infant
Dilated cardiomyopathy is one of the most common causes of the heart failure in childhood and can develop as a consequence of metabolic disorders such as fatty acid beta-oxidation disorders or amino acids metabolism disorders. In our report we present a case where in the course of deficiency of long-chain 3-hydroxyacyl-coenzyme A dehydrogenase the recovery from dilated cardiomyopathy resulted in hypertrophic cardiomyopathy.
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