甲状腺功能减退患者自身免疫性甲状腺炎与缺铁性贫血的关系探讨。临床病例

T. P. Nikolaienko-Kamyshova, E.A. Akhe
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引用次数: 0

摘要

摘要:贫血是一个需要确定的全球公共卫生问题。在确定贫血综合征的检查和治疗算法时,有必要仔细研究其发展的危险因素,并考虑合并症。在几个方向上进行诊断搜索将允许指定贫血综合征的发展机制,在联合恶化临床状况。在内分泌病理背景下出现贫血综合征表现的患者,即甲状腺功能减退中的自身免疫性甲状腺炎,尤其值得关注,因为其在育龄妇女中的发病率明显增加,这决定了本文的研究方向和目的。本文报道1例慢性自身免疫性甲状腺炎伴甲状腺功能减退、子宫腺肌症状态,并伴有子宫异常出血,并发缺铁性贫血的临床观察与治疗。由于患者最初认为子宫出血是主要问题,她被妇科医生观察;同时,建立了有利于贫血的血液综合分析指标的变化。子宫内膜异位症的治疗和铁补充剂的预约导致实验室参数正常化的改善。患者6个月没有咨询医生,没有进行实验室控制。如果她感觉更糟了,她就去看家庭医生。在内分泌学家的参与下进行仔细的检查,可以诊断出甲状腺功能减退状态下的慢性自身免疫性甲状腺炎,并给予适当的治疗,这有助于使患者的病情正常化。结论。贫血综合征的表现需要确定其发生的原因,以及检查和监测治疗的算法的发展。全科医生应该是研究贫血的原因和管理病人在门诊阶段与其他专业的医生的参与主要协调。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
ABOUT CONNECTION AUTOIMMUNE THYROIDITIS WITH MANIFESTATION OF IRON DEFICIENCY ANEMIA IN A CONDITION OF HYPOTHYROIDISM IN GENERAL CLINICAL PRACTICE. CLINICAL CASE
Summary: Anemia is a global public health problem that needs to be identified. When determining the algorithms for examination and treatment of anemic syndrome, it is necessary to carefully study the risk factors for its development, taking into account comorbidity. Carrying out of diagnostic search in several directions will allow to specify mechanisms of development of an anemic syndrome which at a combination worsen a clinical condition. Special attention should be paid to patients with manifestations of anemic syndrome on the background of endocrine pathology, namely autoimmune thyroiditis in hypothyroidism, as the incidence rate among women of childbearing age has increased significantly, which determines the direction of research and aim of this publication. The article presents a clinical case of observation and treatment of a patient with chronic autoimmune thyroiditis in a state of hypothyroidism and adenomyosis, accompanied by abnormal uterine bleeding, which was complicated by iron deficiency anemia. Because the patient initially considered uterine bleeding to be the main problem, she was observed by a gynecologist; at the same time changes of indicators of the general analysis of blood in favor of anemia are established. Treatment of endometriosis and the appointment of iron supplements led to improvement with the normalization of laboratory parameters. For 6 months the patient did not consult a doctor, no laboratory control was performed. If she felt worse, the patient consulted a family doctor. Careful examination with the involvement of an endocrinologist allowed to diagnose chronic autoimmune thyroiditis in a state of hypothyroidism and prescribe adequate therapy, which helped to normalize the patient's condition. Conclusion. Manifestations of anemic syndrome require determining the causes of its occurrence with the development of algorithms for examination and monitoring of treatment. The general practice physician should be the main coordinator in studying the causes of anemia and the management of patients in the outpatient phase with the involvement of physicians of other specialties.
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