covid - 19相关格林-巴利综合征临床病例

U.M. Petrova, Y. Garmash, O. Klimenko, V.V. Bedny, O.G. Mylnikova, V.M. Eagle
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引用次数: 0

摘要

我们的文章旨在揭示COVID-19感染后吉兰-巴利综合征的发展问题材料和方法我们采用的主要研究方法是科学研究和实际描述我们自己的临床病例。采用实验室研究方法、磁共振成像和肌电图确诊并进行鉴别诊断。结果。2021年,急性神经病学和疼痛治疗中心治疗了12名格林-巴利综合征患者。格林-巴利综合征(GBS)是一种急性自身免疫性炎症性多根神经病变。GBS的临床表现包括四肢进行性运动和感觉障碍以及疼痛。我们提请您注意患者B在“Feofania”临床医院接受治疗的一例GBS临床病例。患者B, 32岁,因四肢无力、行走困难、咽喉异物感、吞咽困难、胸部不适、呼吸困难等主诉入住Feofania临床医院。在发病前几周,患者因轻度SARS-CoV-2感染接受了治疗。患者病情在发病第25天逐渐恶化,尽管进行了血浆置换、静脉注射免疫球蛋白和糖皮质激素治疗。然而,从第29天开始,病情逐渐改善,2个月后复查时,患者仍有轻度下肢远端肌肉麻痹(多达4个点),下肢肌腱反射减少。盆腔器官的敏感性和功能未见异常。结论。在我们的文章中,我们报告了一个与covid -19相关的GBS病例,其典型的临床表现,周围性轻瘫加重,稳定,并通过病理治疗和后续康复措施逐渐恢复所有功能(呼吸,盆腔功能,运动,感觉和反射)。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
CLINICAL CASE OF COVID-ASSOCIATED GUILLAIN-BARRE SYNDROME
The aim of our article is to shed light on the problem of the development of Guillain-Barre syndrome after COVID-19 infection Materials and methods The main research methods we used were scientific research and the actual description of our own clinical case. Laboratory research methods, magnetic resonance imaging, and electromyography were used to confirm the diagnosis and perform differential diagnostics. Results. In 2021, the Center for Acute Neurology and Pain Treatment treated 12 patients with Guillain-Barre syndrome. Guillain-Barre syndrome (GBS) is an acute autoimmune inflammatory polyradiculoneuropathy. The clinical picture of GBS includes progressive motor and sensory disorders in the extremities and pain. We bring to your attention a clinical case of GBS in patient B., who was treated at the clinical hospital "Feofania". Patient B., 32 years old, was hospitalized in the Feofania Clinical Hospital with complaints of weakness in her arms and legs, inability to walk, foreign body sensation in the throat, difficulty swallowing, chest discomfort, and difficulty breathing. A few weeks before the disease, the patient was treated for a mild SARS-CoV-2 infection. The patient's condition gradually deteriorated by day 25 of the disease, despite plasmapheresis, intravenous immunoglobulin and glucocorticoid therapy. However, from day 29, the condition gradually improved and on re-examination after 2 months, the patient remained mild distal paresis of the leg muscles (up to 4 points), decreased tendon reflexes from the lower extremities. Disorders of sensitivity and function of the pelvic organs were not observed. Conclusion. In our article, we present a case of COVID-19-associated GBS with a classic clinical picture, increased peripheral paresis, stabilization, and gradual recovery of all functions (respiration, pelvic function, motor, sensory, and reflex) through pathogenetic treatment and follow-up rehabilitation measures.
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CiteScore
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34
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