心脏功能

Revista. Circulo Argentino de Odontologia Pub Date : 2002-01-15 DOI:10.1126/scisignal.1152002tw23

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引用次数: 65

摘要

本周的《自然》杂志有很大一部分是关于心脏病学的。在这些文章中，有几篇是分子性质的，可能会引起STKE读者的兴趣。Bers回顾了控制离子跨膜运输导致心脏高度协调收缩的机制，而Marbán回顾了各种离子通道的突变和慢性修饰如何导致心律失常和心力衰竭。Rockman等人回顾了已知参与心功能的G蛋白偶联受体。心脏兴奋-收缩耦合。自然，415,198-205(2002)。[在线杂志]E. Marbán，心脏通道病变。自然，415,213-218(2002)。[在线杂志]李建军，李建军，李建军，等。跨膜受体与心脏功能的关系。自然，415,206-212(2002)。(在线期刊)

本文章由计算机程序翻译，如有差异，请以英文原文为准。

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Cardiac Function

This week's issue of Nature contains a large section devoted to cardiology. Of those articles, several are of a molecular nature, and may be of interest to STKE readers. Bers reviews the mechanisms that govern the transport of ions across membranes leading to the highly orchestrated contraction of the heart, whereas Marbán reviews how mutations and chronic modifications of various ion channels can lead to cardiac arrhythmias and heart failure. Rockman et al. review the G protein-coupled receptors known to be involved in cardiac function. D. M. Bers, Cardiac excitation-contraction coupling. Nature 415, 198-205 (2002). [Online Journal] E. Marbán, Cardiac channelopathies. Nature 415, 213-218 (2002). [Online Journal] H. A. Rockman, W. J. Koch, R. J. Lefkowitz, Seven-transmembrane-spanning receptors and heart function. Nature 415, 206-212 (2002). [Online Journal]

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来源期刊

Revista. Circulo Argentino de Odontologia

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