II类病灶的仪器评估

G. Battista , L. Guida , P. Avvanzo , M. Mastrovincenzo , C. Chimenti , L. Lo Muzio , D. Ciavarella
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引用次数: 1

摘要

目的探讨咀嚼肌在低发散和高发散角型错颌畸形中可能的病因作用。材料与方法对10例Angleⅱ类错颌畸形患者进行体表肌电图、t -扫描和运动图检查。结果与高发散患者相比,低发散患者表现出所有肌肉的激活增加,尤其是颞前肌和咬肌,它们表现出更前的咬合重心和更快的开合速度。结论下颌骨结构和/或位置改变可能与肌肉活动功能障碍有关。肌肉功能障碍可改变生长模式,尤其是在低发散患者中。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Valutazione strumentale delle malocclusioni di Classe II

Objectives

In the present study, the authors investigated the possible etiological role of the masticatory muscles in the development of Angle class II malocclusion in hypodivergent and hyperdivergent patients.

Materials and methods

Ten patients with Angle class II malocclusion were examined, and each underwent surface electromyography, T-Scanning, and kinesiography.

Results

Compared with the hyperdivergent patients, those who were hypodivergent exhibited increased activation of all the muscles studied, especially the anterior temporalis and masseter, which presented a more anterior occlusal barycenter and higher-velocity opening and closing.

Conclusion

Structural and/or positional changes in the mandibles can be related to dysfunctional muscle activity. The muscle dysfunction can alter the growth pattern, especially in hypodivergent patients.

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