人类心脏的高应变率压缩行为

Khyati Verma, S. Mukherjee, P. Gaur, A. Chawla, R. Malhotra, S. Lalwani
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引用次数: 1

摘要

车祸中发生的胸部损伤在所有致命和非致命伤害中占很大比例。有限元人体模型用于了解心脏等关键器官的损伤机制,以提高碰撞安全性。通过研究其在冲击应变率(高达300/s)下的压缩行为,可以深入了解其损伤机制。本研究报告了在0.001/s至200/s的应变速率下对心脏组织进行的总共20次压缩测试。格林应变是从位移中计算出来的,这些位移是从高速视频记录的分析中获得的。根据测得的力和初始横截面积计算应力。研究表明,心脏组织的反应是非线性的,并且与应变率有关。弹性模量也随应变而变化,在15%至46%的压缩应变下,其值在1.79e-3MPa至3.34MPa之间。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
High strain rate compressive behaviour of human heart
Thoracic injuries incurred during crashes constitute a significant portion of all fatal and non-fatal injuries. Finite element human body models are used to understand the injury mechanisms to critical organs like the heart for improving crash safety. Major insight can be gained into its injury mechanisms by studying its compressive behaviour at strain rates seen in impact (up to 300/s). This study reports a total of 20 compression tests performed on heart tissues at strain rates ranging from 0.001/s to 200/s. Green strain was calculated from displacements which were obtained from analysis of high speed video recordings. Stresses were calculated from the measured force and initial cross-sectional area. The study showed that the response of heart tissue was non-linear and strain rate dependent. The elastic modulus also varied with strain with values ranging from 1.79e-3 MPa to 3.34 MPa at compressive strain of 15% to 46%.
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