低血糖舞蹈病中可逆的透镜状核T1高强度

Q3 Medicine
D. Chouksey, Pankaj Rathi, N. Goyal, A. Sodani
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引用次数: 0

摘要

急性至亚急性发作的舞蹈病通常是由中风、代谢、感染后和自身免疫性疾病引起的。糖尿病(DM)患者的绒毛膜炎主要是由于高血糖引起的,很少有病例是由于低血糖引起的。细胞水平的机制仍在发展。糖尿病舞蹈病患者的MRI变化有不同的报道。重要的是要讨论我们的低血糖性脑脊髓炎患者,因为其独特的特征,如低BMI,最近诊断的磺脲类糖尿病有低血糖发作史,以及用神经抑制剂控制的急性左脑脊髓炎,她的MRI大脑显示可逆的豆状T1核高信号,没有扩散限制,在SWI上开花。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
A reversible lentiform nucleus T1 hyperintensity in hypoglycemic chorea
Acute to subacute onset chorea is commonly due to stroke, metabolic, post-infectious, and autoimmune disorders. Chorea in diabetes (DM) patient is mostly due to hyperglycemia, and few cases were reported due to hypoglycemia. The mechanism at the cellular level is still evolving. The MRI changes in diabetic chorea patients are variably reported. It is important to discuss our patient with hypoglycemic hemichorea because of unique features like low BMI, recently diagnosed DM on sulphonylurea with a history of hypoglycemic episodes, and acute left hemichorea that was controlled with neuroleptics, and her MRI brain showed reversible lentiform nucleus T1 hyperintensity without diffusion restriction and blooming on SWI.
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来源期刊
Annals of Movement Disorders
Annals of Movement Disorders Medicine-Surgery
CiteScore
0.60
自引率
0.00%
发文量
0
审稿时长
17 weeks
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