肾移植后不能恢复正常的微生物组。

IF 6.7 2区 医学 Q1 MEDICINE, RESEARCH & EXPERIMENTAL
Hannah Craven, Helen Erlandsson, Dagmara McGuiness, David McGuiness, Denise Mafra, Umer Ijaz, Peter Bergman, Paul G Shiels, Peter Stenvinkel
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引用次数: 0

摘要

透析和肾移植(Ktx)减轻了慢性肾脏疾病(CKD)的一些生理缺陷,但这些是否减轻了微生物失调和炎症微生物代谢产物的产生,这对尿毒症表型有重要贡献,还有待确定。我们研究了CKD患者和接受KTx的患者循环中存在的细菌DNA特征。随着CKD的进展,我们的数据与日益严重的微生态失调一致,伴随着产生三甲胺(TMA)的致病菌假单胞菌和芽孢杆菌的增加。值得注意的是,与CKD5患者相比,KTx患者表现出显著不同的微生物群,这令人惊讶地包括产生TMA的芽孢杆菌的进一步增加和致致敬乳酸杆菌的损失。KTx后,只有两个属(Viellonella和Saccharimonidales)的丰度存在显著差异,这可能反映了TMA生产者和利用者之间的互惠关系,这取代了标准微生物组的恢复。我们的元数据分析证实,TMA N-氧化物和单碳代谢对炎症负担和微生物组的组成都有显著影响。这表明,这些代谢产物是形成尿毒症微生物组的关键,并可能在制定饮食干预策略时被利用,以减轻CKD的生理缺陷,并使更具致敏性的微生物组得以恢复。
本文章由计算机程序翻译,如有差异,请以英文原文为准。

A normative microbiome is not restored following kidney transplantation.

A normative microbiome is not restored following kidney transplantation.

A normative microbiome is not restored following kidney transplantation.

A normative microbiome is not restored following kidney transplantation.

Dialysis and kidney transplantation (Ktx) mitigate some of the physiological deficits in chronic kidney disease (CKD), but it remains to be determined if these mitigate microbial dysbiosis and the production of inflammatory microbial metabolites, which contribute significantly to the uraemic phenotype. We have investigated bacterial DNA signatures present in the circulation of CKD patients and those receiving a KTx. Our data are consistent with increasing dysbiosis as CKD progresses, with an accompanying increase in trimethylamine (TMA) producing pathobionts Pseudomonas and Bacillus. Notably, KTx patients displayed a significantly different microbiota compared with CKD5 patients, which surprisingly included further increase in TMA producing Bacillus and loss of salutogenic Lactobacilli. Only two genera (Viellonella and Saccharimonidales) showed significant differences in abundance following KTx that may reflect a reciprocal relationship between TMA producers and utilisers, which supersedes restoration of a normative microbiome. Our metadata analysis confirmed that TMA N-oxide (TMAO) along with one carbon metabolism had significant impact upon both inflammatory burden and the composition of the microbiome. This indicates that these metabolites are key to shaping the uraemic microbiome and might be exploited in the development of dietary intervention strategies to both mitigate the physiological deficits in CKD and enable the restoration of a more salutogenic microbiome.

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来源期刊
Clinical science
Clinical science 医学-医学:研究与实验
CiteScore
11.40
自引率
0.00%
发文量
189
审稿时长
4-8 weeks
期刊介绍: Translating molecular bioscience and experimental research into medical insights, Clinical Science offers multi-disciplinary coverage and clinical perspectives to advance human health. Its international Editorial Board is charged with selecting peer-reviewed original papers of the highest scientific merit covering the broad spectrum of biomedical specialities including, although not exclusively: Cardiovascular system Cerebrovascular system Gastrointestinal tract and liver Genomic medicine Infection and immunity Inflammation Oncology Metabolism Endocrinology and nutrition Nephrology Circulation Respiratory system Vascular biology Molecular pathology.
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