炎症性疾病中的内质网应激和氧化应激。

IF 2.6 4区生物学 Q3 BIOCHEMISTRY & MOLECULAR BIOLOGY

DNA and cell biology Pub Date : 2022-11-01 Epub Date: 2022-09-14 DOI:10.1089/dna.2022.0353

Yun Tang, Xiangping Zhou, Ting Cao, En Chen, Yumeng Li, Wenbo Lei, Yibao Hu, Bisha He, Shuangquan Liu

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引用次数: 9

摘要

内质网(ER)应激和氧化应激(OS)通常是细胞的相关状态，是正常生理的一部分，但更频繁地表现在许多疾病的病理生理中，特别是涉及急性或慢性炎症的疾病。在这项研究中，我们回顾了最近关于内质网应激和OS在炎症性疾病发病机制中的作用的研究成果。

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Endoplasmic Reticulum Stress and Oxidative Stress in Inflammatory Diseases.

Endoplasmic reticulum (ER) stress and oxidative stress (OS) are often related states in cells as part of normal physiology but more frequently manifested in the pathophysiology of many diseases, particularly diseases involving acute or chronic inflammation. In this study, we reviewed recent findings about the role of ER stress and OS in the pathogenesis of inflammatory diseases.

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来源期刊

DNA and cell biology 生物-生化与分子生物学

CiteScore

6.60

自引率

0.00%

发文量

审稿时长

1.5 months

期刊介绍： DNA and Cell Biology delivers authoritative, peer-reviewed research on all aspects of molecular and cellular biology, with a unique focus on combining mechanistic and clinical studies to drive the field forward. DNA and Cell Biology coverage includes: Gene Structure, Function, and Regulation Gene regulation Molecular mechanisms of cell activation Mechanisms of transcriptional, translational, or epigenetic control of gene expression Molecular Medicine Molecular pathogenesis Genetic approaches to cancer and autoimmune diseases Translational studies in cell and molecular biology Cellular Organelles Autophagy Apoptosis P bodies Peroxisosomes Protein Biosynthesis and Degradation Regulation of protein synthesis Post-translational modifications Control of degradation Cell-Autonomous Inflammation and Host Cell Response to Infection Responses to cytokines and other physiological mediators Evasive pathways of pathogens.