继发于非动脉瘤性蛛网膜下腔出血的孤立性动眼神经麻痹。

Shyle Mehta, Abhijith Bathini, Anwesha Dubey, Awinita Barpujari, Ahmad Kassem, Mohanad Sulaiman, Mandy Binning
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引用次数: 2

摘要

我们报告了两例在非动脉瘤性SAH (NASAH)后发展为单侧颅神经III (CNIII)麻痹的患者。蛛网膜下腔出血(SAH)可表现为多种体征和症状。早期诊断对确定疗程至关重要。因此,临床医生必须意识到这种情况的可变临床表现。两名患者因SAH入住同一机构。患者1,男,52岁,主要表现为头痛,左眼上睑下垂,无痛性复视。非对比头部计算机断层扫描(CT)显示左侧脑裂内有SAH,中脑和镰叶周围有血液。患者2,70岁男性,表现为轻度头痛,急性视力模糊,右眼上睑下垂。非对比头部CT显示弥漫性SAH主要位于鞍上池和鞍上池。患者被送进神经重症监护室,接受血管造影诊断以确定可能的动脉瘤。两例患者的磁共振和血管造影均为阴性。患者接受最佳药物治疗,并在门诊进行随访。两例患者均在NASAH背景下发现单侧CNIII性麻痹。诊断性血管造影未见动脉瘤;因此,SAH被确定为自发的。我们认为单侧CNIII麻痹可能是NASAH的征兆。
本文章由计算机程序翻译,如有差异,请以英文原文为准。

Isolated oculomotor nerve palsy secondary to non-aneurysmal subarachnoid hemorrhage.

Isolated oculomotor nerve palsy secondary to non-aneurysmal subarachnoid hemorrhage.

Isolated oculomotor nerve palsy secondary to non-aneurysmal subarachnoid hemorrhage.

Isolated oculomotor nerve palsy secondary to non-aneurysmal subarachnoid hemorrhage.

We present a case series of two patients who developed unilateral cranial nerve III (CNIII) palsy following non-aneurysmal SAH (NASAH). Subarachnoid hemorrhage (SAH) can present with various signs and symptoms. Early diagnosis is paramount to determine treatment course. Thus, clinicians must be aware of the variable clinical presentations of this condition. Two patients were admitted to a single institution for SAH. Patient 1, 52-year-old male, presented with headache, left eye ptosis, and painless diplopia. A non-contrast head computed tomography (CT) demonstrated a SAH within the left sylvian fissure and blood surrounding the mesencephalon and falx. Patient 2, 70-year-old male, presented with mild headache, acute onset of blurry vision, and right eye ptosis. A non-contrast head CT demonstrated a diffuse SAH predominantly in the Sylvian and suprasellar cisterns. Patients were admitted to the neuro intensive care unit and underwent diagnostic angiograms to identify possible aneurysms. Magnetic resonance imaging and angiograms for both patients were negative. Patients were managed with best medical therapy and followed up in the outpatient setting. Unilateral CNIII palsy in the setting of NASAH was identified in both patients. Diagnostic angiograms were negative for aneurysms; therefore, SAH were determined to be spontaneous. We propose that unilateral CNIII palsy is a possible sign of NASAH.

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