中枕区与恶意与善意创造力的差异:近红外光谱研究。

IF 1.7 4区 医学 Q4 NEUROSCIENCES
Social Neuroscience Pub Date : 2022-04-01 Epub Date: 2022-02-10 DOI:10.1080/17470919.2022.2038261
Xinuo Qiao, Kelong Lu, Jing Teng, Zhenni Gao, Ning Hao
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引用次数: 5

摘要

本研究旨在利用功能近红外光谱(fNIRS)研究恶意创造(MC)过程中潜在想法产生的神经相关因素。参与者被要求在三种类型的创造力任务中解决问题:恶意创造力任务(MCT)、善意创造力任务(BCT)和替代用途任务(AUT)。fNIRS用于记录任务过程中个体的大脑活动。结果显示,在MCT期间,被试右侧枕中区(rMO)的神经激活较弱,右侧额极皮质(rFPC)与rMO之间的神经耦合(NC)较低。这些结果表明,rfp和rMO之间的r-MO活性和NC可以区分恶意和善意的创造性思维形式。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Middle occipital area differentially associates with malevolent versus benevolent creativity: An fNIRS investigation.

This study aimed to explore the neural correlates underlying idea generation during malevolent creativity (MC) using functional near-infrared spectroscopy (fNIRS). Participants were asked to solve problems during three types of creativity tasks: malevolent creativity task (MCT), benevolent creativity task (BCT), and alternative uses task (AUT). fNIRS was used to record individual cerebral activity during the tasks. The results revealed that participants demonstrated weaker neural activation in the right middle occipital area (rMO) and lower neural coupling (NC) between the right frontopolar cortex (rFPC) and rMO during MCT than during BCT and AUT. These suggest that r-MO activity and NC between the rFPC and rMO may distinguish between malevolent and benevolent forms of creative ideation.

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来源期刊
Social Neuroscience
Social Neuroscience 医学-神经科学
CiteScore
3.40
自引率
5.00%
发文量
36
审稿时长
6-12 weeks
期刊介绍: Social Neuroscience features original empirical Research Papers as well as targeted Reviews, Commentaries and Fast Track Brief Reports that examine how the brain mediates social behavior, social cognition, social interactions and relationships, group social dynamics, and related topics that deal with social/interpersonal psychology and neurobiology. Multi-paper symposia and special topic issues are organized and presented regularly as well. The goal of Social Neuroscience is to provide a place to publish empirical articles that intend to further our understanding of the neural mechanisms contributing to the development and maintenance of social behaviors, or to understanding how these mechanisms are disrupted in clinical disorders.
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