杂合POLG变异Ser1181Asn在常染色体显性轴突神经病、近端肌肉疲劳、上睑下垂和红色纤维粗糙的家族中共分离。

Maike F Dohrn, Corina Heller, Diana Zengeler, Carolin D Obermaier, Saskia Biskup, Joachim Weis, Stefan Nikolin, Kristl G Claeys, Ulrike Schöne, Danique Beijer, Natalie Winter, Pascal Achenbach, Burkhard Gess, Jörg B Schulz, Lejla Mulahasanovic
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引用次数: 3

摘要

通过全外显子组测序,发现POLG杂合变异c.3542G>A;p.Ser1181Asn在一个四人患病家庭中,表现为混合性神经肌病表型。该变异位于聚合酶γ的活性位点,位于与常染色体显性遗传相关的群集区域。在青春期,该指数发展为远端萎缩和无力,感觉丧失,传入共济失调,复视和双侧上睑下垂。其中一个姐姐表现出沙科-玛丽牙样症状,而最小的妹妹和父亲报告了运动引起的肌肉疼痛和近端无力。在这些个体中,我们没有观察到中枢神经系统受到任何影响。父亲和姐姐的肌肉活组织检查显示纤维呈红色,电镜检查证实线粒体受损。我们得出结论,这种新的POLG变异解释了这个家族的表型。
本文章由计算机程序翻译,如有差异,请以英文原文为准。

Heterozygous POLG variant Ser1181Asn co-segregating in a family with autosomal dominant axonal neuropathy, proximal muscle fatigability, ptosis, and ragged red fibers.

Heterozygous POLG variant Ser1181Asn co-segregating in a family with autosomal dominant axonal neuropathy, proximal muscle fatigability, ptosis, and ragged red fibers.

By whole-exome sequencing, we found the heterozygous POLG variant c.3542G>A; p.Ser1181Asn in a family of four affected individuals, presenting with a mixed neuro-myopathic phenotype. The variant is located within the active site of polymerase gamma, in a cluster region associated with an autosomal dominant inheritance. In adolescence, the index developed distal atrophies and weakness, sensory loss, afferent ataxia, double vision, and bilateral ptosis. One older sister presented with Charcot-Marie-Tooth-like symptoms, while the youngest sister and father reported exercise-induced muscle pain and proximal weakness. In none of the individuals, we observed any involvement of the central nervous system. Muscle biopsies obtained from the father and the older sister showed ragged-red fibers, and electron microscopy confirmed mitochondrial damage. We conclude that this novel POLG variant explains this family's phenotype.

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