遗传性痉挛性截瘫导致无法复原的马足,双侧跟腱延长后步态能力的改善:1例报告。

Jorik Nonnekes, Noël Keijsers, Angelique Witteveen, Alexander Geurts
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引用次数: 1

摘要

目的:在遗传性痉挛性截瘫患者中,由于小腿肌肉进行性缩短而导致脚趾行走是常见的。跟腱延长是一种治疗选择,但临床医生通常不愿使用这种方法,因为它可能导致小腿肌肉减弱,随后在站立步态阶段踝关节力量减弱和膝关节不稳定。我们在这里报告一个病例报告,支持这些负面的副作用可以避免在精心挑选的人遗传性痉挛性截瘫。方法:对一名29岁无并发症的遗传性痉挛性截瘫女性患者进行双侧跟腱延长,并联合双侧胫骨后肌和趾屈肌肌腱切断术,该患者由于比目鱼肌和腓骨肌缩短而出现进行性步态不稳定(导致马蹄形无法复位)。结果:双侧跟腱延长导致主观和客观结果的改善。自选步态速度由术前的0.75 m/s提高到术后的1.07 m/s (p < 0.001)。站立期的膝关节不稳定在术后未发生。术后踝关节力矩轨迹恢复正常,踝关节峰值力量增加。结论:选择遗传性痉挛性截瘫患者,采用跟腱延长矫治双侧不可复位马足可改善其步态能力。
本文章由计算机程序翻译,如有差异,请以英文原文为准。

Improved Gait Capacity after Bilateral Achilles Tendon Lengthening for Irreducible Pes Equinus Due to Hereditary Spastic Paraplegia: a Case Report.

Improved Gait Capacity after Bilateral Achilles Tendon Lengthening for Irreducible Pes Equinus Due to Hereditary Spastic Paraplegia: a Case Report.

Improved Gait Capacity after Bilateral Achilles Tendon Lengthening for Irreducible Pes Equinus Due to Hereditary Spastic Paraplegia: a Case Report.

Improved Gait Capacity after Bilateral Achilles Tendon Lengthening for Irreducible Pes Equinus Due to Hereditary Spastic Paraplegia: a Case Report.

Objective: Toe walking due to progressive shortening of the calf muscles is common in people with hereditary spastic paraplegia. Achilles tendon lengthening is a treatment option, but clinicians are often hesitant to use this procedure, as it may result in weakening of the calf muscles and, subsequently, in reduced ankle power and knee instability during the stance phase of gait. We report here a case report supporting that these negative side-effects can be avoided in well-selected people with hereditary spastic paraplegia.

Method: Bilateral Achilles tendon lengthening, combined with bilateral tenotomy of the tibialis posterior and toe flexors, was performed in a 29-year-old woman with uncomplicated hereditary spastic paraplegia who experienced progressive gait instability due to shortening of the soleus and gastrocnemius muscles (resulting in irreducible pes equinus).

Results: Bilateral Achilles tendon lengthening resulted in improvement in both subjective and objective outcomes. Self-selected gait speed improved from 0.75 m/s before surgery to 1.07 m/s after surgery (p < 0.001). Knee instability during the stance phase did not occur post-surgery. The ankle moment trajectories normalized after surgery, while peak ankle powers increased.

Conclusion: Correction of bilateral irreducible pes equinus by Achilles tendon lengthening may improve gait capacity in well-selected subjects with hereditary spastic paraplegia.

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