先前鼻给药安塔哥米-122可预防辐射引起的脑损伤。

Haihong Zhou, Furong Sun, Mingqian Ou, Yu Zhang, Meijun Lin, Liqin Song, Yangsheng Yu, Haojie Liao, Weihao Fan, Huaijie Xing, Minhua Li, Kui Zhao, Xiaolian Wu, Yuanhong Sun, Chunmei Liang, Yujie Cai, Lili Cui
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引用次数: 6

摘要

放射性脑损伤是头颈部肿瘤治疗的主要不良事件,影响50%以上接受放射治疗并长期生存的患者的生活质量。然而,这些患者没有有效的治疗方法,必须开发预防性药物和有效的给药方法。根据我们的研究结果,miR-122-5p在小鼠辐射性脑损伤(RBI)模型和接受放射治疗的鼻咽癌(NPC)患者中表达上调。照射前单次鼻内给药antagomiR-122-5p可有效减轻RBI模型小鼠辐射诱导的认知障碍、神经元损伤和神经炎症。结果进一步表明,在小胶质细胞模型中,miR-122-5p抑制可降低促炎细胞因子水平,增强吞噬功能,从而保护细胞免受辐射诱导的神经元损伤。此外,我们分析了转录组数据并验证了Tensin 1 (TNS1)可能是miR-122-5p在RBI中的靶标。总之,我们的研究结果揭示了miR-122-5p在RBI中调节神经炎症的独特作用,表明鼻内给药miR-122-5p的无创策略可能是RBI中一个有吸引力的治疗靶点,为临床试验提供了新的见解。进一步进行系统的安全性评价,优化给药方式,明确作用机制,将加快其进入临床的进程。
本文章由计算机程序翻译,如有差异,请以英文原文为准。

Prior nasal delivery of antagomiR-122 prevents radiation-induced brain injury.

Prior nasal delivery of antagomiR-122 prevents radiation-induced brain injury.

Prior nasal delivery of antagomiR-122 prevents radiation-induced brain injury.

Radiation-induced brain injury is a major adverse event in head and neck tumor treatment, influencing the quality of life for the more than 50% of patients who undergo radiation therapy and experience long-term survival. However, no effective treatments are available for these patients, and preventative drugs and effective drug-delivery methods must be developed. Based on our results, miR-122-5p was upregulated in the mouse radiation-induced brain injury (RBI) model and patients with nasopharyngeal carcinoma (NPC) who received radiation therapy. Intranasal administration of a single antagomiR-122-5p dose before irradiation effectively alleviated radiation-induced cognitive impairment, neuronal injury, and neuroinflammation in the mouse RBI model. Results further indicated that miR-122-5p inhibition in microglia reduced the levels of proinflammatory cytokines and enhanced the phagocytic function to protect against radiation-induced neuronal injury in cell models. Further, we profiled transcriptome data and verified that Tensin 1 (TNS1) may be the target of miR-122-5p in RBI. In summary, our results reveal a distinct role for miR-122-5p in regulating neuroinflammation in RBI, indicating that a non-invasive strategy for intranasal miR-122-5p administration may be an attractive therapeutic target in RBI, providing new insights for clinical trials. Further systematic safety assessment, optimization of drug administration, and clarity of mechanism will accelerate the process into clinical practice.

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