Myt1表达失调是重度抑郁症和双相情感障碍的潜在外周生物标志物。

IF 1.8 4区 医学 Q3 GENETICS & HEREDITY
Journal of neurogenetics Pub Date : 2021-09-01 Epub Date: 2021-05-20 DOI:10.1080/01677063.2021.1928663
Maryam Ghanbarirad, Mehrdad Hashemi, Seyed Mehdi Saberi, Ahmad Majd
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引用次数: 0

摘要

重度抑郁症(MDD)和双相情感障碍(BPD)是最使人衰弱的精神疾病。重度抑郁症的诊断标准包括心理和生理症状,如情绪低落、食欲或睡眠改变。BPD除了抑郁期外,还表现为躁狂或轻躁狂的发作,情绪和能量水平的升高与这种情况有关。成人神经发生和髓鞘形成失调在精神疾病中也有报道。作为神经发生的关键因素,Myt1基因的表达可能在这些情况下发生改变。采用Real-time PCR技术,对100例MDD患者和100例BPD患者的Myt1表达水平进行了评估,并与健康对照(HC)进行了比较。结果显示Myt1在MDD和BPD中显著下调。Logistic回归分析和二元分类评价分别揭示了Myt1的潜在危险因素和生物标志物特征。此外,前向和后向数字广度结果显示MDD和BPD受试者的工作记忆功能显著降低。相关分析显示Myt1下调与WM破坏之间存在显著关联。总之,由于其在神经发生中的作用改变,Myt1的下调可能与MDD和BPD的病理有关。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Dysregulation of Myt1 expression acts as a potential peripheral biomarker for major depressive disorder and bipolar disorder.

Major depressive disorder (MDD) and bipolar disorder (BPD) are among the most debilitating mental conditions. Diagnostic criteria for MDD include psychological and physical symptoms, such as low mood and changes in appetite or sleep, respectively. BPD in addition to periods of depression represents episodes of mania or hypomania, and elevation in mood and energy levels are associated with this condition. Dysregulation in adult neurogenesis and myelination have been reported in psychiatric disorders. As a key factor in neurogenesis, it was hypothesized that Myt1 gene expression may be altered in these conditions. Using Real-time PCR, Myt1 expression level in 100 MDD patients and 100 BPD patients, compared with healthy control (HC) individuals was evaluated. Results demonstrate significant downregulation of Myt1 in MDD and BPD. Logistic regression analysis and binary classification evaluation reveal potential risk factor and biomarker characteristics of Myt1, respectively. Moreover, forward and backward digit span results denote a significant reduction in the function of working memory (WM) of MDD and BPD subjects. Correlation analysis revealed a significant association between Myt1 downregulation and WM disruption in the affected individuals. In conclusion, due to its altered role in neurogenesis, downregulation of Myt1 can be associated with the pathology of MDD and BPD.

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来源期刊
Journal of neurogenetics
Journal of neurogenetics 医学-神经科学
CiteScore
4.40
自引率
0.00%
发文量
13
审稿时长
>12 weeks
期刊介绍: The Journal is appropriate for papers on behavioral, biochemical, or cellular aspects of neural function, plasticity, aging or disease. In addition to analyses in the traditional genetic-model organisms, C. elegans, Drosophila, mouse and the zebrafish, the Journal encourages submission of neurogenetic investigations performed in organisms not easily amenable to experimental genetics. Such investigations might, for instance, describe behavioral differences deriving from genetic variation within a species, or report human disease studies that provide exceptional insights into biological mechanisms
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