将 "促多巴胺调节(KB220Z™)"作为克服美国鸦片/类鸦片流行病静息状态多巴胺张力降低的长期治疗方法。

K Blum, F Marcelo, K Dushaj, L Fried, R D Badgaiyan
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引用次数: 0

摘要

众所周知,复发、道德问题和住院治疗与多巴胺 D2 受体 A1 等位基因的存在有关,而这种基因变异的携带者倾向于氨基酸疗法,因此,直观地说,应将平衡或恢复多巴胺功能减退的方法视为克服当前美国鸦片剂/阿片类药物流行病的前线策略,使数百万人免于死亡和不必要的成瘾锁定。如果我们继续沿着以毒攻毒的原始道路走下去,我们注定会失败。这一教训也具有全球意义。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
"Pro-dopamine regulation (KB220Z™)" as a long-term therapeutic modality to overcome reduced resting state dopamine tone in opiate/opioid epidemic in America.

Since it is known that relapse, morality, and hospitalizations have been tied to the presence of the Dopamine D2 Receptor A1 allele, as one example, and carriers of this gene variant have a proclivity to favor amino-acid therapy, it seems intuitive that the incorporation of modalities to provide a balance and or restoration of hypodopaminergia should be considered as a front-line tactic to overcome the current American opiate/opioid epidemic, saving millions from death and unwanted locked-in-addiction. If we continue down the prim road path of fighting addiction to narcotics with narcotics, we are doomed to fail. This lesson can also have global interest.

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