Shikha Jain, K Sadashiva Shetty, Anaveri T Prakash, Mamta Agrawal, Shweta Jain
{"title":"永久性下颌犬牙嵌塞:扩展我们的理解。","authors":"Shikha Jain, K Sadashiva Shetty, Anaveri T Prakash, Mamta Agrawal, Shweta Jain","doi":"","DOIUrl":null,"url":null,"abstract":"<p><strong>Introduction: </strong>Eruption disturbances, tooth size and specific malocclusions are known to be genetically influenced. The clinical association between these traits may indicate common genetic controls.</p><p><strong>Objectives: </strong>A cross-sectional clinical study was designed to test the null hypothesis that the maximum mesiodistal crown diameter (MMD) of maxillary and mandibular central and lateral incisors and the prevalence of various classes of incisor relationships (Class I, II/1, II/2 and III) do not differ between the subjects with and without permanent mandibular canine(s) impaction.</p><p><strong>Methods: </strong>Dental models of 43 subjects diagnosed with mandibular canine(s) impaction (Impaction Group - IG) were compared with those of 86 subjects of a control reference sample (Control Group - CG). Independent t-test and chi-square tests were used to determine the association between mandibular canine(s) impaction and the MMD of the incisors and the incisor relationship, respectively. The likelihood of various incisor relationships between the IG and CG were evaluated according to odds ratios.</p><p><strong>Results: </strong>A fourfold increase (p < 0.0001) in the overall frequency of Class II/2 incisor relationship was observed in the IG when compared to controls.</p><p><strong>Conclusions: </strong>The null hypothesis was rejected. Subjects with mandibular canine(s) impaction appeared to be characterised with wider incisors and a remarkably high rate of Class II/2 malocclusion. This information assists the understanding of genetically controlled dental anomalies, which are likely to coexist with mandibular canine(s) impaction.</p>","PeriodicalId":55417,"journal":{"name":"Australian Orthodontic Journal","volume":"30 1","pages":"39-44"},"PeriodicalIF":0.0000,"publicationDate":"2014-05-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":"{\"title\":\"Permanent mandibular canine(s) impaction: expansion of our understanding.\",\"authors\":\"Shikha Jain, K Sadashiva Shetty, Anaveri T Prakash, Mamta Agrawal, Shweta Jain\",\"doi\":\"\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"<p><strong>Introduction: </strong>Eruption disturbances, tooth size and specific malocclusions are known to be genetically influenced. The clinical association between these traits may indicate common genetic controls.</p><p><strong>Objectives: </strong>A cross-sectional clinical study was designed to test the null hypothesis that the maximum mesiodistal crown diameter (MMD) of maxillary and mandibular central and lateral incisors and the prevalence of various classes of incisor relationships (Class I, II/1, II/2 and III) do not differ between the subjects with and without permanent mandibular canine(s) impaction.</p><p><strong>Methods: </strong>Dental models of 43 subjects diagnosed with mandibular canine(s) impaction (Impaction Group - IG) were compared with those of 86 subjects of a control reference sample (Control Group - CG). Independent t-test and chi-square tests were used to determine the association between mandibular canine(s) impaction and the MMD of the incisors and the incisor relationship, respectively. The likelihood of various incisor relationships between the IG and CG were evaluated according to odds ratios.</p><p><strong>Results: </strong>A fourfold increase (p < 0.0001) in the overall frequency of Class II/2 incisor relationship was observed in the IG when compared to controls.</p><p><strong>Conclusions: </strong>The null hypothesis was rejected. Subjects with mandibular canine(s) impaction appeared to be characterised with wider incisors and a remarkably high rate of Class II/2 malocclusion. This information assists the understanding of genetically controlled dental anomalies, which are likely to coexist with mandibular canine(s) impaction.</p>\",\"PeriodicalId\":55417,\"journal\":{\"name\":\"Australian Orthodontic Journal\",\"volume\":\"30 1\",\"pages\":\"39-44\"},\"PeriodicalIF\":0.0000,\"publicationDate\":\"2014-05-01\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Australian Orthodontic Journal\",\"FirstCategoryId\":\"1085\",\"ListUrlMain\":\"\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"Q\",\"JCRName\":\"Medicine\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Australian Orthodontic Journal","FirstCategoryId":"1085","ListUrlMain":"","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"Q","JCRName":"Medicine","Score":null,"Total":0}
引用次数: 0
摘要
出牙障碍,牙齿大小和特定的错咬合是已知的遗传影响。这些特征之间的临床联系可能表明共同的遗传控制。目的:一项横断面临床研究旨在检验零假设,即上颌和下颌中切牙和侧切牙的最大中远端冠直径(MMD)以及各种类型的切牙关系(I类,II/1类,II/2类和III类)的患病率在有和没有永久性下颌犬牙嵌塞的受试者之间没有差异。方法:将43例诊断为下颌犬牙嵌塞(impaction Group - IG)的受试者与86例对照组(control Group - CG)的模型进行比较。分别采用独立t检验和卡方检验确定下颌犬牙嵌塞与门牙MMD及门牙关系之间的关系。根据比值比评估IG和CG之间各种切牙关系的可能性。结果:与对照组相比,IG中II类/2类切牙关系的总频率增加了4倍(p < 0.0001)。结论:零假设被否定。下颌犬齿嵌塞患者的特点是门牙较宽,II/2类错牙合发生率高。这些信息有助于理解遗传控制的牙齿异常,这些异常可能与下颌犬牙嵌塞共存。
Permanent mandibular canine(s) impaction: expansion of our understanding.
Introduction: Eruption disturbances, tooth size and specific malocclusions are known to be genetically influenced. The clinical association between these traits may indicate common genetic controls.
Objectives: A cross-sectional clinical study was designed to test the null hypothesis that the maximum mesiodistal crown diameter (MMD) of maxillary and mandibular central and lateral incisors and the prevalence of various classes of incisor relationships (Class I, II/1, II/2 and III) do not differ between the subjects with and without permanent mandibular canine(s) impaction.
Methods: Dental models of 43 subjects diagnosed with mandibular canine(s) impaction (Impaction Group - IG) were compared with those of 86 subjects of a control reference sample (Control Group - CG). Independent t-test and chi-square tests were used to determine the association between mandibular canine(s) impaction and the MMD of the incisors and the incisor relationship, respectively. The likelihood of various incisor relationships between the IG and CG were evaluated according to odds ratios.
Results: A fourfold increase (p < 0.0001) in the overall frequency of Class II/2 incisor relationship was observed in the IG when compared to controls.
Conclusions: The null hypothesis was rejected. Subjects with mandibular canine(s) impaction appeared to be characterised with wider incisors and a remarkably high rate of Class II/2 malocclusion. This information assists the understanding of genetically controlled dental anomalies, which are likely to coexist with mandibular canine(s) impaction.