原发性胆汁性肝硬化中胆道上皮细胞凋亡、自噬和衰老。

Hepatitis research and treatment Pub Date : 2010-01-01 Epub Date: 2010-11-04 DOI:10.1155/2010/205128
Motoko Sasaki, Yasuni Nakanuma
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引用次数: 13

摘要

原发性胆汁性肝硬化(PBC)是一种慢性胆汁淤积性肝病,其血清学特征是抗线粒体自身抗体(AMAs)的高发,组织学特征是小胆管胆管炎,最终导致小胆管广泛丧失。临床和实验研究均提示其自身免疫性发病机制,但其病因、ama在胆管病变发病中的意义等方面仍存在争议。胆道上皮细胞(BECs)凋亡的独特特性,其中自身抗原暴露于免疫系统的效应物,被认为是PBC胆管病变的一个原因。近年来的研究表明,细胞衰老和自噬参与了胆管病变的发生。衰老的BECs可能通过表达衰老相关的分泌表型(包括各种趋化因子)来调节管周微环境,并参与PBC胆管病变的发病机制。
本文章由计算机程序翻译,如有差异,请以英文原文为准。

Biliary epithelial apoptosis, autophagy, and senescence in primary biliary cirrhosis.

Biliary epithelial apoptosis, autophagy, and senescence in primary biliary cirrhosis.

Biliary epithelial apoptosis, autophagy, and senescence in primary biliary cirrhosis.

Biliary epithelial apoptosis, autophagy, and senescence in primary biliary cirrhosis.

Primary biliary cirrhosis (PBC) is a chronic cholestatic liver disease characterized serologically by the high prevalence of anti-mitochondrial autoantibodies (AMAs) and histologically by the cholangitis of small bile ducts, eventually followed by extensive loss of the small bile duct. An autoimmune pathogenesis is suggested by clinical and experimental studies, but there remain issues regarding the etiology, the significance of AMAs in the pathogenesis of bile duct lesions, and so on. The unique properties of apoptosis in biliary epithelial cells (BECs), in which there is exposure of autoantigen to the effectors of the immune system, are proposed to be a cause of bile duct lesions in PBC. Recent progress disclosed that cellular senescence and autophagy are involved in bile duct lesions in PBC. Senescent BECs may modulate the periductal microenvironment by expressing senescence-associated secretory phenotypes, including various chemokines, and contribute to the pathogenesis of bile duct lesions in PBC.

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