不同创伤和物质依赖史个体的情绪反应。

Alicia K Klanecky, Dennis E McChargue
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引用次数: 8

摘要

背景:研究报告了未发展为PTSD (TWP)的创伤个体的物质依赖率很高。虽然过去的研究未能始终如一地证明TWP个体会出现PTSD症状,但研究结果表明,TWP和尼古丁依赖(SDH)以外的物质依赖史与影响中断有关。目的:本研究探讨了四组不同SDH和TWP的积极和消极情感机制,包括TWP + SDH,仅TWP,仅SDH或无病史。研究人员假设,随着TWP和SDH共存的增加,与中性情绪诱导相比,成年人(n = 78)对实验诱导的消极情绪的情绪反应会更强烈。方法:经过简短的电话筛选,符合条件的参与者完成基线自我报告问卷和实验操纵的消极和中性情绪诱导。结果:最值得注意的是,结果显示TWP与SDH与情绪诱导的相互作用显著(F (1,63) = 4.154;Mse = 51.999;P = .046)。简单效应表明,除了TWP + SDH个体外,所有参与者在消极情绪条件下的积极情绪都比中性情绪条件下显著减少。结论:研究结果可能确定TWP和SDH病史患者复发的保护机制。
本文章由计算机程序翻译,如有差异,请以英文原文为准。

Emotional reactivity across individuals with varying trauma and substance dependence histories.

Emotional reactivity across individuals with varying trauma and substance dependence histories.

BACKGROUND: Research has reported a high rate of substance dependence in traumatized individuals who do not develop PTSD (TWP). While past studies have failed to consistently demonstrate that TWP individuals experience PTSD symptoms, findings have indicated that TWP and a history of substance dependence aside from nicotine dependence (SDH) are linked to affect disruption. AIMS: The present study explored positive and negative affective mechanisms across four groups with varying SDH and TWP including TWP + SDH, TWP only, SDH only, or no history. Researchers hypothesized that adults (n = 78) would be more emotionally reactive to an experimentally-induced negative mood compared to a neutral mood induction as the presence of co-existing TWP and SDH increased. METHOD: After a brief telephone screening, eligible participants completed baseline self-report questionnaires and experimentally-manipulated negative and neutral mood inductions. RESULTS: Most notably, results showed a significant TWP x SDH x Mood induction interaction (F (1, 63) = 4.154; Mse = 51.999; p = .046) for positive affect responses. Simple effects indicated that all participants except TWP + SDH individuals experienced a significant decrease in positive affect during the negative compared to the neutral mood condition. CONCLUSION: Findings may identify a protective mechanism for relapse among individuals with a history of both TWP and SDH.

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