子宫骶后悬吊神经病的病因学。

M A Schön Ybarra, R E Gutman, D Rini, V L Handa
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引用次数: 7

摘要

前言和假设:我们研究的目的是寻找子宫骶韧带悬吊(USLS)后所报告的神经性病变的神经学解释[2-3]。方法:我们解剖了7具经防腐处理的女性成年尸体的左韧带下神经结构。我们进行了文献回顾,以确定每个暴露的神经结构的脊神经感觉纤维组成和它所支配的皮节。然后,我们比较了预期的每种神经结构的感觉神经病变与USLS后的神经病变,以确定哪种神经结构卡压可以解释报告的症状。结果:子宫骶韧带(USL)下的几个神经结构在USLS中很容易被缝线夹住。讨论了夹闭的预期临床结果。结论:骶神经丛第二干或坐骨神经盆腔内部分的S2感觉纤维卡压是USLS后报道的神经病变最合理的病因。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Etiology of post-uterosacral suspension neuropathies.

Introduction and hypothesis: The goal of our investigation was to find a neurological explanation for neuropathies reported following some uterosacral ligament suspension (USLS) [2-3].

Methods: We dissected the neural structures beneath the USL in seven female, adult, embalmed cadavers. We made a literature review to determine the spinal nerve sensory fiber composition of each exposed neural structure and the dermatome(s) that it innervates. We then compared anticipated sensory neuropathies for each neural structure with neuropathies following USLS to determine which neural structure entrapment could explain the reported symptoms.

Results: Several neural structures located beneath the uterosacral ligament (USL) are vulnerable to suture entrapment during USLS. Anticipated clinical outcomes of entrapments are discussed.

Conclusions: Entrapment of S2 sensory fibers in the second trunk of the sacral plexus or in the intrapelvic portion of the sciatic nerve is the most plausible etiology for reported neuropathies following USLS.

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