病毒和非病毒介导的Nk4传递到肿瘤的抗转移作用。

Alexandra Buhles, Sara A Collins, Jan P van Pijkeren, Simon Rajendran, Michelle Miles, Gerald C O'Sullivan, Deirdre M O'Hanlon, Mark Tangney
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引用次数: 23

摘要

癌症患者最常见的死亡原因是癌症转移。肿瘤细胞的转移行为受细胞外生长因子调控,如肝细胞生长因子(HGF), c-Met受体酪氨酸激酶的配体,c-Met受体的异常表达/激活与转移进展密切相关。Nk4(也称为白介素(IL)32b)是HGF c-Met系统的竞争性拮抗剂,可抑制c-Met信号传导和肿瘤转移。Nk4具有独立于hgf拮抗剂功能的额外抗血管生成活性。抑制血管生成以及癌症特异性诱导细胞凋亡的作用使Nk4序列成为癌症基因治疗的一个有吸引力的候选者。本研究探讨了基因治疗介导原发肿瘤产生Nk4对肿瘤转移的抑制作用。为了达到最高的治疗效果,抗癌基因的最佳递送至关重要。非病毒质粒传递方法具有安全性和易于生产的优点,尽管在大多数肿瘤中短暂存在,但可以立即进行转基因表达。抗血管生成分子的持续存在是抗血管生成治疗的首选,而腺相关病毒(AAV)介导的长期表达可能是这方面更合适的递送方式。然而,AAV载体达到适当的基因表达水平所需的孵育时间阻碍了在许多快速生长的小鼠肿瘤模型中的效果。在这里,我们描述了小鼠试验,评估了Nk4通过质粒脂肪转染或AAV2载体传递到原发肿瘤时对自发转移性刘易斯肺癌(LLC)模型的影响。瘤内给药AAV-Nk4产生了最高的治疗反应,原发性肿瘤生长和肺转移发生率均显著降低。质粒介导的治疗也能显著减少转移性肿瘤的生长,但对原发性皮下肿瘤的生长有中等程度的减少。总的来说,本研究表明,当通过AAV或非病毒方法传递时,Nk4基因治疗转移性肿瘤的潜力。
本文章由计算机程序翻译,如有差异,请以英文原文为准。

Anti-metastatic effects of viral and non-viral mediated Nk4 delivery to tumours.

Anti-metastatic effects of viral and non-viral mediated Nk4 delivery to tumours.

Anti-metastatic effects of viral and non-viral mediated Nk4 delivery to tumours.

Anti-metastatic effects of viral and non-viral mediated Nk4 delivery to tumours.

The most common cause of death of cancer sufferers is through the occurrence of metastases. The metastatic behaviour of tumour cells is regulated by extracellular growth factors such as hepatocyte growth factor (HGF), a ligand for the c-Met receptor tyrosine kinase, and aberrant expression/activation of the c-Met receptor is closely associated with metastatic progression. Nk4 (also known as Interleukin (IL)32b) is a competitive antagonist of the HGF c-Met system and inhibits c-Met signalling and tumour metastasis. Nk4 has an additional anti-angiogenic activity independent of its HGF-antagonist function. Angiogenesis-inhibitory as well as cancer-specific apoptosis inducing effects make the Nk4 sequence an attractive candidate for gene therapy of cancer. This study investigates the inhibition of tumour metastasis by gene therapy mediated production of Nk4 by the primary tumour. Optimal delivery of anti-cancer genes is vital in order to achieve the highest therapeutic responses. Non-viral plasmid delivery methods have the advantage of safety and ease of production, providing immediate transgene expression, albeit short-lived in most tumours. Sustained presence of anti-angiogenic molecules is preferable with anti-angiogenic therapies, and the long-term expression mediated by Adeno-associated Virus (AAV) might represent a more appropriate delivery in this respect. However, the incubation time required by AAV vectors to reach appropriate gene expression levels hampers efficacy in many fast-growing murine tumour models. Here, we describe murine trials assessing the effects of Nk4 on the spontaneously metastatic Lewis Lung Carcinoma (LLC) model when delivered to primary tumour via plasmid lipofection or AAV2 vector. Intratumoural AAV-Nk4 administration produced the highest therapeutic response with significant reduction in both primary tumour growth and incidence of lung metastases. Plasmid-mediated therapy also significantly reduced metastatic growth, but with moderate reduction in primary subcutaneous tumour growth. Overall, this study demonstrates the potential for Nk4 gene therapy of metastatic tumours, when delivered by AAV or non-viral methods.

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