一氧化氮在乙烯诱导蚕豆气孔关闭中的作用[j]。

植物生理与分子生物学学报 Pub Date : 2007-08-01
Jie Li, Li-Yan Qiu, Fang-Gui Zhao, Li-Xia Hou, Xin Liu
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引用次数: 0

摘要

研究了一氧化氮(NO)和乙烯对蚕豆气孔运动的影响。结果表明,NO供体SNP(硝普钠)10微mol/L和乙烯0.04%均能显著诱导气孔关闭,且两者共同作用时均能促进气孔关闭。AVG(一种乙烯合成抑制剂)、c-PTIO(一种特定的NO清除剂)和NaN(一种NR抑制剂)处理后,NO-和乙烯诱导的气孔关闭作用被抑制,而一氧化氮合酶(NOS)抑制剂的作用几乎没有。我们推测NO和乙烯在气孔关闭调控中有协同作用;乙烯可能通过调节硝酸还原酶(NR)依赖性NO的产生来诱导气孔关闭。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
[The role of nitric oxide in ethylene-induced stomatal closure in Vicia faba L].

The effects of nitric oxide (NO) and ethylene on Vicia faba L. stomatal movement were studied. The results showed that NO donor SNP (sodium nitroprusside) 10 micromol/L and ethylene 0.04% could induce stomatal closure distinctly and they could promote stomatal closure when treated together. When treated with AVG (an inhibitor of ethylene synthesis), c-PTIO (a specific scavenger of NO) and NaN(3) (an inhibitor of NR), the effects of NO- and ethylene-induced stomatal closure were inhibited but the inhibitor of nitric oxide synthase (NOS) had little effect. We presumed that there was coordinative effect between NO and ethylene in regulation of stomatal closure; ethylene could induce stomatal closure by regulating the production of nitrate reductase (NR)-dependent NO.

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