应激源抑制主动脉平滑肌收缩功能和反应性的机制。

Fiziologicheskii zhurnal imeni I.M. Sechenova Pub Date : 1996-07-01

A P Solodkov, A P Bozhko

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引用次数: 0

摘要

阻断NO合成并没有改变应激源对乙酰胆碱诱导的内皮依赖性松弛的增强。固定应激似乎抑制了收缩功能和主动脉平滑肌反应性，这是内皮细胞中一氧化氮基础生成增加的结果。

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[The mechanism of stressor suppression of the contractile function and reactivity of aortic smooth muscle].

Blockade of the NO synthesis did not change the stressor augmentation of the acetylcholine-induced endothelium-dependent relaxation. The immobilisation stress seems to suppress the contractile function and the reactivity of the aorta smooth muscle reactivity as the result of augmentation of the NO basal production in endotheliocytes.

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Fiziologicheskii zhurnal imeni I.M. Sechenova

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